Chronic Paroxysmal Hemicrania: Heart Rate Changes and ECG Rhythm Disturbances. A Computerized Analysis of 24 h Ambulatory ECG Recordings

Russell, David; Storstein, Liv

doi:10.1046/j.1468-2982.1984.0402135.x

Cited by 20 publications

(11 citation statements)

References 19 publications

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“…Bradycardia could be formidable, decreasing to 20 beats per minute [32]. In our case, less drastic irregularity was present (and in a later series) [33]. These phenomena could possibly fit with a cholinergic stimulation, less marked in CPH than in cluster headache.…”

Section: Cardiac Dysrhythmiasupporting

confidence: 43%

Chronic paroxysmal hemicrania: From the index patient to the disease

Sjaastad

2006

Current Science Inc

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The first patient with chronic paroxysmal hemicrania (CPH), a 41-year-old woman, first seen in 1961, was followed until an adequate treatment was found, 12 years later. Clinically, attack frequency and duration differed widely from the general pattern of cluster headache. Ocular variables, such as intraocular pressure and corneal indentation pulse amplitudes, also differed in our case (clear symptomatic side increment during attacks) and cluster headache. Pupil reactions to directly and indirectly acting sympathicomimetic drugs were also vastly different in our case and cluster headache: no signs of Horner s syndrome in our patient, while cluster headache exhibits a "Horner-like pattern." In cluster headache, there is a relative hypohidrosis in the forehead on the symptomatic side if body temperature is increased, and a clear hyperhidrosis on direct parasympathomimetic stimulation. This was not so in our case. Indomethacin was highly effective in our case, while "cluster headache drugs," such as ergotamine/sumatriptan, were ineffective. Indomethacin was inactive in cluster headache. Accordingly, our case seemed to differ decisively from cluster headache: CPH had been discovered.

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Section: Cardiac Dysrhythmiasupporting

confidence: 43%

Chronic paroxysmal hemicrania: From the index patient to the disease

Sjaastad

2006

Current Science Inc

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“…Almost all migraine patients have nausea or vomiting; many of them also have other gastrointestinal symptoms, e.g. [7][8][9][10][11][12][13][14][15][16][17][18] As a result of plasma catecholamine determinations, it has been suggested that patients with migraine show sympathetic hypofunction, the defective noradrenergic activity being an important pathogenic factor, 15 but quite opposite results have also been reported. Extraordinary sweating can be encountered during migraine attacks.…”

Section: Introductionmentioning

confidence: 99%

Cardiovascular Reflexes in Young Migraine Patients

Havanka‐Kanniainen¹,

Tolonen²,

Myllylä³

1986

Headache

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SYNOPSIS The autonomic nervous system functions of 49 young migraine patients, aged from 11 to 22 years, wereinvestigated during headache‐free intervals by utilizing a number of cardiovascular reflexes as indices.Twenty‐five healthy subjects of similar age served as a control group. The pulse rate (R‐R‐interval)variation was registered in normal and deep breathing, during the Valsalva manoeuvre, and in anorthostatic test (a tilt table was used); and the blood pressure measurements were performed in anorthostatic test and during an isometric work test. There were no significant differences between the groupof young migraine patients and the control group. Furthermore, all numeric mean values were in thenormal limits given in the literature; only incidental abnormal values were encountered, and even then inboth groups. Moreover, the cardiovascular reflex responses of various subgroups of migraine patients(common‐classic, frequent ‐infrequent attacks, patients with or without autonomic nervous systemdysfunction signs during attacks, normal‐abnormal EEGs), did not significantly differ from each otherduring this interictal recording period. The results suggest intact autonomic nervous system functions inyoung migraine patients during headache free intervals.

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“…Autonomic studies have yielded inconsistent results, although increased lacrimation and nasal secretion on the symptomatic side and decreased salivation has been reported [99]. Cardiac conduction abnormalities, including bradycardia and sinoatrial block, bundle branch block with runs of atrial fibrillation, extrasystoles, and bradycardia, have occurred in patients with CPH [43,61,100].…”

Section: Laboratory Studiesmentioning

confidence: 99%

Effective management of ice pick pains, SUNCT, and episodic and chronic paroxysmal hemicrania

Newman

2001

Current Science Inc

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Idiopathic stabbing headaches, the SUNCT syndrome, and the paroxysmal hemicranias are a group of primary headache disorders that are characterized by brief, short-lived attacks of head pain, which recur multiple times throughout the day. These syndromes are much less prevalent than other types of primary headaches such as migraine and tension-type headaches but are significantly more disabling. Recognition of these uncommon disorders is important because their management differs from standard headache therapies.

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Chronic Paroxysmal Hemicrania: Heart Rate Changes and ECG Rhythm Disturbances. A Computerized Analysis of 24 h Ambulatory ECG Recordings

Cited by 20 publications

References 19 publications

Chronic paroxysmal hemicrania: From the index patient to the disease

Chronic paroxysmal hemicrania: From the index patient to the disease

Cardiovascular Reflexes in Young Migraine Patients

Effective management of ice pick pains, SUNCT, and episodic and chronic paroxysmal hemicrania

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