Chronic respiratory morbidity after prolonged and premature rupture of the membranes.

Thompson, Peter J.; Greenough, Anne; Nicolaides, Kypros H.

doi:10.1136/adc.65.8.878

Cited by 14 publications

(3 citation statements)

References 18 publications

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“…Persistent respiratory morbidity has also been described in infants born after prolonged premature rupture of the membranes during pregnancy 12. The infants reported here were not severely ill in the neonatal period and showed good “catch up” lung growth in relation to body weight during the first year of life to around the lower end of the normal range of values.…”

Section: Discussionsupporting

confidence: 62%

Lesson of the Week: Pulmonary hypoplasia presenting as persistent tachypnoea in the first few months of life

Aiton

Fox

Hannam

et al. 1996

BMJ

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Section: Discussionsupporting

confidence: 62%

Lesson of the Week: Pulmonary hypoplasia presenting as persistent tachypnoea in the first few months of life

Aiton

Fox

Hannam

et al. 1996

BMJ

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“…The radiographic appearances are variable from patchy consolidation to areas of collapse with intervening overinflation (Fig. 21) [12].…”

Section: Infectionmentioning

confidence: 99%

The neonatal chest

Shaw

1997

Eur Radiol

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Introduction Neonatal respiratory distress and the chest radiographRespiratory distress in the newborn infant commonly results from disease in the respiratory and cardiovascular systems. However, pathology in other organ systems may be reflected in cardiorespiratory compromise. Clinical and biochemical evaluation can be complemented by simple plain radiography in many cases, although resorting to more sophisticated techniques is helpful in more difficult cases [1]. In this review the causes of respiratory distress are discussed in relation to the various organ systems in which pathology is manifest.

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“…There is a spectrum of severity of pulmonary hypoplasia. Some survivors may require many months of supplementary oxygen and recurrent respiratory symptoms are common [8], but infants with mild pulmonary hypoplasia only suffer a raised respiratory rate in infancy [9]. It is important, therefore, that pulmonary hypoplasia and the severity of the condition is accurately diagnosed antenatally to enable practitioners and parents to make informed decisions regarding whether termination of the pregnancy or in utero intervention is appropriate for their *Address correspondence to this author at the Regional Neonatal Intensive Care Centre, 4 th Floor Golden Jubilee Wing, King's College Hospital, Denmark Hill, London, SE5 9RS, USA; Tel: 020 7346 3037; Fax: 020 7346 8284; E-mail: anne.greenough@kcl.ac.uk baby.…”

Section: Introductionmentioning

confidence: 99%

Prediction and Prevention of Abnormal Antenatal Lung Growth

Prendergast¹,

Greenough

2007

CRMR

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Pulmonary hypoplasia, the result of abnormal antenatal lung growth, occurs as a primary anomaly or more usually the consequence of disorders resulting in reduction of amniotic fluid volume, intra-thoracic space or fetal breathing movements. Two dimensional (2D) ultrasound examination yields useful information on amniotic fluid volume and the presence of congenital abnormalities associated with pulmonary hypoplasia. Assessment of the thoracic or lung size by 2D ultrasound examination, is insufficiently specific, but a very low lung to head ratio predicts a fatal outcome in fetuses with congenital diaphragmatic hernia (CDH). Three-D ultrasound examination provides information on fetal lung volume prior to 34 weeks of gestation but can be inaccurate if there is oligohydramnios. Magnetic resonance imaging is expensive and has limited patient acceptability. Antenatal interventions attempted to prevent pulmonary hypoplasia include amnioinfusion, which can facilitate ultrasound examination, but only temporarily relieves oligohydramnios. Thoraco-amniotic shunting results in effective drainage of pleural effusions, facilitating resuscitation, but is usually performed too late in pregnancy to influence lung growth. In utero surgical repair of CDH has been attempted, but a more promising technique is obstruction of the normal egress of fetal lung fluid by placing a balloon in the trachea.

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Chronic respiratory morbidity after prolonged and premature rupture of the membranes.

Cited by 14 publications

References 18 publications

Lesson of the Week: Pulmonary hypoplasia presenting as persistent tachypnoea in the first few months of life

Lesson of the Week: Pulmonary hypoplasia presenting as persistent tachypnoea in the first few months of life

The neonatal chest

Prediction and Prevention of Abnormal Antenatal Lung Growth

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