Circulation coronaire et métabolisme du myocarde dans le cœur pulmonaire chronique

Moret, Pierre; Bopp, P; Hatam, Kassem; Panos, J

doi:10.1159/000168922

Cited by 4 publications

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“…It has been reported that mean LDL levels are higher and HDL levels are lower than controls in children who are diagnosed with epilepsy and use carbamazepine (10). Interestingly, serum lipids were low in patients with chronic obstructive pulmonary disease, and it was determined as a cause of low cardiovascular disease in these patients (11,12). In our study, TC and LDL values of our patient group were found in the normal percentile values, while TG and HDL levels were found above the percentile values compared to normal controls.…”

Section: Discussionsupporting

confidence: 45%

Factors affecting lipid profile in pediatric palliative care

Harputluoğlu¹

2022

Erciyes Med J

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Section: Discussionsupporting

confidence: 45%

Factors affecting lipid profile in pediatric palliative care

Harputluoğlu¹

2022

Erciyes Med J

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“…The results were comparable with those in healthy subjects, or somewhat lower (Moret, Bopp, Hatam & Panos, 1966;Hosono, 1965;Rose & Hoffman, 1954). In view of the arterial hypoxaemia in COPD, it is probable that 'normal' values of coronary blood flow are not sufficient to satisfy the myocardial oxygen uptake, particularly under conditions of increased work load.…”

Section: Low Coronary Blood $Owmentioning

confidence: 57%

Left Ventricular Function in Chronic Obstructive Pulmonary Disease with and without Cardiac Failure

Jezek

Schrijen

1973

Clinical Science

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1. The circulatory response to supine exercise and to angiotensin infusion was measured in patients with chronic obstructive pulmonary disease (COPD).2. Left ventricular function assessed from left ventricular end-diastolic pressure (LVEDPFstroke volume or stroke work relationships was almost always normal in the patients without cardiac failure. The patients with right heart failure showed an abnormal response with a pronounced rise of LVEDP during both exercise and infusion of angiotensin. The values of LVEDP showed a significant correlation with RVEDP and with arterial oxygen saturation and carbon dioxide tension under both experimental conditions. No abnormality in the response of cardiac output, mean systolic ejection ratio or systolic ejection time was observed. In thirteen out of fifteen patients subjected to both exercise and infusion of angiotensin, the results of the two tests agreed in showing left ventricular function to be normal or abnormal; two patients showed an abnormal response to exercise, but a normal response to angiotension.3. The possible causes of the abnormality of left ventricular function in COPD with right heart failure are discussed. Changes in left ventricular compliance and the effect of chronic hypoxaemia and hypercapnic acidosis on myocardial contractility seem to be the most probable explanation.

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Comparative Effects of Exercise and Isocapnic Voluntary Hyperventilation on Pulmonary Haemodynamics in Chronic Bronchitis and Emphysema

Lockhart¹,

Nader²,

Tzareva³

et al. 1970

Eur J Clin Investigation

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(1) Twelve patients with chronic bronchitis and/or pulmonary emphysema with borderline pulmonary artery Eressure and arterial blood gases have been submitted to aemodynamic studies in order to compare changes in the pulmonary circulation brought about by leg exercise and by voluntary hyperventilation with comparable breathing rate, tidal volume and arterial blood gases during both periods. (2) Minute ventilation (Exercise: 12.1 ± 1.9 l. min‐l m‐2; hyperventilation: 12.8± 1.8 1 · min‐1· m‐2), breathing rate (E: 22.6 ±3.4, H: 23.5 ±3.8), percentage arterial oxygen saturation (E: 94.7±3.3%, H: 95.9±2.7%) and carbon dioxide tension (E: 45.5 ± 5.1 mm Hg, H: 46.8 ± 5.1 mm Hg) did not differ significantly during exercise and hyperventilation, thus suggesting that both mechanical forces acting upon intrathoracic structures and pulmonary vasomotor tone were comparable. (3) During exercise pulmonary arterial mean pressure (31.4±5.7mm Hg) and pulmonary wedge pressure (13.2± 4.2 mm Hg) rose significantly (p < 0.001) and consistently. During hyperventilation pulmonary arterial mean pressure rose slightly (22.1 ± 5.5 mm Hg, p < 0.05) and the increase in average pulmonary wedge pressure was insignificant (10.0±5.4 mm Hg), although the latter rose markedly in five cases. Pulmonary vascular resistances were not consistently modified. Pulmonary wedge pressure was not correlated with minute ventilation but was positively correlated with the amplitude of respiratory variations in intrathoracic pressure. (4) Thus, our data suggested that the rise in pulmonary artery and wedge pressures during exercise was due to the combined effects of large intrathoracic pressure swings and of an increase in the haemodynamic load of the left ventricle.

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Circulation coronaire et métabolisme du myocarde dans le cœur pulmonaire chronique

Cited by 4 publications

References 0 publications

Factors affecting lipid profile in pediatric palliative care

Factors affecting lipid profile in pediatric palliative care

Left Ventricular Function in Chronic Obstructive Pulmonary Disease with and without Cardiac Failure

Comparative Effects of Exercise and Isocapnic Voluntary Hyperventilation on Pulmonary Haemodynamics in Chronic Bronchitis and Emphysema

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