Clinical and electrophysiologic characteristics of left septal atrial tachycardia

Marrouche, Nassir F.; SippensGroenewegen, A.; Yang, Yanfei; Dibs, Samer; Scheinman, Melvin M.

doi:10.1016/s0735-1097(02)02071-5

Cited by 78 publications

(76 citation statements)

References 23 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…[1][2][3][4]8 Regarding the sensitivity of adenosine and the mechanism of focal AT, Markowitz et al 15 proposed that adenosine sensitive focal AT is commonly due to triggered activity or automaticity; however, their adenosine-insensitive focal reentrant ATs showed low-amplitude, fractionated electrograms at the site of origin, which occupy a large percent of tachycardia cycle length, 15 different from the ATs presented in this study or those reported previously. [1][2][3][4]8 In addition, Horie et al 16 reported 7 cases of adenosine-sensitive focal reentrant ATs originating from the proximal coronary sinus. Although fractionated electrograms occupying a large percentage of tachycardia cycle length were not observed in their cases, a single extrastimulus delivered from the slow pathway region, distant from the EAAS, reset the AT with a postpacing interval identical to the AT cycle length.…”

Section: Mechanism Of At and Its Substratecontrasting

confidence: 62%

Analysis of the Anatomical Tachycardia Circuit in Verapamil-Sensitive Atrial Tachycardia Originating From the Vicinity of the Atrioventricular Node

Yamabe

Tanaka

Morihisa

et al. 2010

Circ: Arrhythmia and Electrophysiology

View full text Add to dashboard Cite

Background-Calcium channel-dependent tissue has been suggested to be involved in the circuit of verapamil-sensitive atrial tachycardia originating from the atrioventricular (AV) node vicinity (V-AT), but little information exists. Methods and Results-To examine the tachycardia circuit of V-AT, a single extrastimulus was delivered during tachycardia to 10 sites of the intraatrial septum: the earliest atrial activation site; His bundle (HB) site; 3 arbitrarily divided sites on the AV junction extending from the HB site to the coronary sinus ostium (CSOS) (sites S, M, and I); the internal-CSOS, inferior-CSOS, superior-CSOS, posterior-CSOS, and posteroinferior-CSOS in 10 patients with V-AT. The longest coupling interval that reset V-AT and subsequent return cycle were measured. The longest coupling interval at earliest atrial activation site was significantly longer than the longest coupling interval at the HB site, site S, M, and I, internal-CSOS, inferior-CSOS, superior-CSOS, posterior-CSOS, and posteroinferior-CSOS, respectively (PϽ0.001 for HB site and PϽ0.0001 for the remaining 8 sites). The return cycle at earliest atrial activation site did not differ from the tachycardia cycle length, whereas those at the remaining 9 sites were significantly longer than tachycardia cycle length (PϽ0.001). Furthermore, a single extrastimulus delivered from sites inferior to the HB site advanced His potential without resetting V-AT in 7 patients in whom AV block was not observed during tachycardia. Conclusions-Atrial tissue within the Koch's triangle extending from the HB site to posteroinferior-CSOS is not involved in the tachycardia circuit. Verapamil-sensitive atrial tissue close to the AV node but not the AV nodal conducting system forms the tachycardia circuit of V-AT. (Circ Arrhythm Electrophysiol. 2010;3:54-62.)

show abstract

Section: Mechanism Of At and Its Substratecontrasting

confidence: 62%

Analysis of the Anatomical Tachycardia Circuit in Verapamil-Sensitive Atrial Tachycardia Originating From the Vicinity of the Atrioventricular Node

Yamabe

Tanaka

Morihisa

et al. 2010

Circ: Arrhythmia and Electrophysiology

View full text Add to dashboard Cite

show abstract

“…Tachycardia mechanism: Reports regarding focal left ATs arising from the MAAo junction are sparse [1][2][3][4] and their mechanisms are unclear. In previous reports, the underlying mechanisms of similar ATs were considered to be microreentry 4) or triggered activity.…”

Section: Discussionmentioning

confidence: 99%

“…In previous reports, the underlying mechanisms of similar ATs were considered to be microreentry 4) or triggered activity. 3) In earlier experimental studies, 6,7) the MA-Ao junction was found to be arrhythmogenic with respect to atrial arrhythmias due to catecholamine-induced delayed afterdepolarization.…”

Section: Discussionmentioning

confidence: 99%

Successful Catheter Ablation of Focal Left Atrial Tachycardia Originating From the Mitral Annulus Aorta Junction

et al. 2006

View full text Add to dashboard Cite

SUMMARYFocal left atrial tachycardias (AT) originating from the mitral annulus-aorta (MA-Ao) junction are rare and their mechanisms are unclear. We report a 35-year-old male with successful ablation of an exercise-induced focal AT due to triggered activity originating from the MA-Ao junction. The AT occurred spontaneously during treadmill exercise testing and was easily induced by an atrial extrastimulus and atrial burst pacing after intravenous administration of isoproterenol. The AT was terminated by an atrial extrastimulus as well as a bolus of 5 mg of adenosine 5'-triphosphate. The coupling intervals of the extrastimuli that induced the AT were positively correlated with the interval between the extrastimuli and the first beat of the AT, suggesting the triggered activity as a tachycardia mechanism. The AT was successfully eliminated by a focal ablation at the MA-Ao junction with the earliest atrial activation where fractionated atrial potentials were recorded. The MA-Ao junction should be recognized as an important arrhythmogenic region. (Int Heart J 2006; 47: 461-468)

show abstract

“…In the left atrium, focal tachycardias were localized to mitral annulus, roof, posterior wall, appendage and septum (Dong J et al, 2005). Left septal atrial tachycardia is rare and the electroanatomic mapping facilitates its ablation (Marrouche NF et al, 2002). Atrial tachycardia originating in the mitral annulus is also rare and the electro-anatomic mapping also facilitates its ablation (Kistler PM et al, 2003).…”

Section: Left Atrial Tachycardiamentioning

confidence: 99%

“…Interestingly, the number of publication describing the use of electroanatomic mapping for ablation of focal tachycardia ablation is much larger (Kottkamp H et al, 1997;Marchlinski FE et al, 1998;Cummings RM et al, 2008). There is also ample of information on the use of this mapping in the ablation of different left atrial tachycardias (Iwai S et al, 2002;Marrouche NF et al, 2002;Kistler PM et al, 2003;Dong J et al, 2005). Left atrial tachycardias may originate on the septum, along the mitral annulus and from the pulmonary veins.…”

Section: Atrial Tachycardia Ablationmentioning

confidence: 99%

Electromagnetic Mapping During Complex RF Ablations

Rosenheck¹,

Banker²,

Weiss³

et al. 2012

Cardiac Arrhythmias - New Considerations

View full text Add to dashboard Cite

Clinical and electrophysiologic characteristics of left septal atrial tachycardia

Abstract: Left septal AT ablation is safe and effective. There was no consistent P-wave morphology associated with this particular type of AT. This arrhythmia appears to be resistant to adenosine and moderately responsive to calcium antagonists.

Cited by 78 publications

References 23 publications

Analysis of the Anatomical Tachycardia Circuit in Verapamil-Sensitive Atrial Tachycardia Originating From the Vicinity of the Atrioventricular Node

Analysis of the Anatomical Tachycardia Circuit in Verapamil-Sensitive Atrial Tachycardia Originating From the Vicinity of the Atrioventricular Node

Successful Catheter Ablation of Focal Left Atrial Tachycardia Originating From the Mitral Annulus Aorta Junction

Electromagnetic Mapping During Complex RF Ablations

Contact Info

Product

Resources

About