Coexistence of Behçet’s Disease and Ankylosing Spondylitis

Küçükşen, Sami; Bağçacı, Sinan; Karahan, Ali Yavuz; Şahin, Muhammed; Uğurlu, Hatice

doi:10.29333/ejgm/82290

Cited by 3 publications

(2 citation statements)

References 10 publications

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“…A Pseudo R-Square value of 11.4% was obtained, meaning that the arm repetitive movement factor had an 11.4% effect on tennis elbow in badminton players, and other factors had 88.6% influence. In line with the research of Karahan et al, (2013), the incidence of tennis elbow in badminton players is only 12.5%, meaning that tennis elbow is a minor injury to badminton players. However, functional activities in badminton players are entirely disrupted, such as when holding the racket and doing backhand movements, because these activities can activate the appearance of pain and reduce the player hitting performance (Buchanan BK, 2022).…”

Section: Discussionsupporting

confidence: 66%

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Contributions of Repetitive Movements to Tennis Elbow Risk in Badminton Players

Grafita,

Rahman

2023

J. pendidik. jasmani olahraga

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Regular practice of badminton players will lead to the formation of repetitive movement patterns as a significant factor causing outer elbow pain, commonly referred as tennis elbow. This study was aimed to identify the contribution of repetitive movements to tennis elbow risks in badminton players, to examine the relationship between repetitive movements and tennis elbow risks, and to investigate the differences of tennis elbow risk based on the repetitive movement status and the player status. The method used was the observational study with a correlational approach involving 122 players of student badminton organization, known as UKM, from four universities selected using the purposive and cluster sampling methods. The dependent variable of this study was the tennis elbow risk, while the independent variables were repetitive movements and the player status. The ordinal linear regression statistical test results obtained a p-value estimated parameter of p = 0.418 and a pseudo R-Square value of 11.4%. Spearman statistical test was obtained (p=0.020) with a value of r = -0.210. The Kruskal-Wallis H statistical test was obtained (p=0.040). The Mann-Whitney statistical test was also obtained (p=0.005). Repetitive movements contribute to the risk of tennis elbow in badminton players and affect the condition. In addition, there is a weak relationship between repetitive movements and tennis elbow. Repetitive movement status and player status affect the risk of tennis elbow in badminton players.

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Section: Discussionsupporting

confidence: 66%

“…The incidence rate of tennis elbow in the productive age of 20-65 years shows the highest risk at 40-50 years (Keijsers et al 2019). In addition, the incidence of tennis elbow is also often found in badminton players, with a prevalence of 12.5% (Karahan et al 2013).…”

Section: Introductionmentioning

confidence: 99%

Contributions of Repetitive Movements to Tennis Elbow Risk in Badminton Players

Grafita,

Rahman

2023

J. pendidik. jasmani olahraga

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Investigation of a possible extended risk haplotype in the IL23R region associated with ankylosing spondylitis

et al. 2017

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The IL23R region on chromosome 1 exhibits complex associations with ankylosing spondylitis (AS). We used publicly available epigenomic information and historical genetic association data to identify a putative regulatory element (PRE) in the intergenic region between IL23R and IL12RB2, which includes two single-nucleotide polymorphisms (SNPs) independently associated with AS—rs924080 (P=2 × 10−3) and rs11578380 (P=2 × 10−4). In luciferase reporter assays, this PRE showed silencer activity (P<0.001). Haplotype and conditional analysis of 4230 historical AS cases and 9700 controls revealed a possible AS-associated extended haplotype, including the PRE and risk variants at three SNPs (rs11209026, rs11209032 and rs924080), but excluding the rs11578380 risk variant. However, the rs924080 association was absent after conditioning on the primary association with rs11209032, which, in contrast, was robust to conditioning on all other AS-associated SNPs in this region (P<2 × 10−8). The role of this putative silencer on some IL23R extended haplotypes therefore remains unclear.

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