“…Maladaptive ER strategies (e.g., suppression, rumination, self-blame, catastrophizing), commonly assessed using the Cognitive Emotion Regulation Reduced ACC and LPFC activation during ECA in anxiety [84]; reduced DLPFC and increased amygdala activation in depression [85] Findings in psychosis-spectrum samples Behavioral Suppression associated with more severe paranoia [87], auditory hallucinations [88], and poorer social functioning [94] Impaired ECA associated with paranoia [99], disorganized symptoms [100], and reduced positive affect [108] Neuroimaging Reduced frontal-limbic connectivity during reappraisal in schizophrenia [103] and PP [104] Reduced LPFC, medial PFC, and ACC activation during ECA in schizophrenia [105] and PP [104] ACC anterior cingulate cortex, BPD borderline personality disorder, CERQ Cognitive Emotion Regulation Questionnaire, DLPFC dorsolateral prefrontal cortex, ECA emotion conflict adaptation, ER emotion regulation, LPFC lateral prefrontal cortex, PFC prefrontal cortex, PP psychosis proneness Questionnaire (CERQ) [75], are increasingly recognized as vulnerability factors that contribute to the development and maintenance of psychopathology [28], particularly disorders characterized by affective instability, such as anxiety [76], depression [77,78], and substance use [79]. Similarly, impaired performance on ECA paradigms requiring inhibition of irrelevant negative emotional stimuli has been observed in depression [80], anxiety [81], borderline personality disorder [82], and those at risk for developing a mood disorder [83].…”