Color Doppler sonography in the diagnosis and monitoring of arterial complications after liver transplantation

Hepatic Arterial Stenosis Assessed with Doppler US after Liver Transplantation: Frequent False-Positive Diagnoses with Tardus Parvus Waveform and Value of Adding Optimal Peak Systolic Velocity Cutoff

“…In approximately 30% of the patients with a falsepositive diagnosis based on the tardus parvus pattern, however, we could not identify any cause. Although a jet fl ow with a PSV greater than 200 cm/sec at the hepatic arterial anastomosis or the juxtaanastomotic part is suggestive of hepatic arterial stenosis ( 2,9,16,17 ), it is often diffi cult to obtain a Doppler signal from a hepatic arterial anastomosis, or it may be diffi cult to identify an anastomosis during the early postoperative period because of a poor sonic window ( 2,9,18 ). In contrast, while the blood fl ow velocity must increase initially to maintain the continuity of volume fl ow as the diameter of the stenotic vessel decreases, once a critical stenosis is reached, any further decrease in stenotic diameter will rapidly reduce the blood fl ow velocity as well as the blood volume fl ow ( 19 ).…”

Laparoscopic treatment of splenic artery aneurysms

“…An RI lower than 0.30 and a SAT Ն 80 ms were considered indicators of insufficient arterial perfusion. 11,12 Power Doppler sonography was also used for signal enhancement and detection of slow flow.…”

The radiologist’s role in managing paediatric liver transplantation: personal experience with 40 patients

“…or because of a less stable clinical condition. In particular, the earliest possible diagnosis of hepatic artery thrombosis, one of the most feared complications due to the high mortality rates and the frequent need for immediate retransplantation, is a determinant for prognosis [24][25][26][27][28]. Clinical and laboratory signs of hepatic artery thrombosis not only are completely nonspecific but tend to develop late, when the transplanted liver has been irreversibly damaged.…”

“…This finding is mostly related to a difference in calibre between the donor's and the receiver's vessels, to a transient oedematous reaction (as confirmed by the gradual spontaneous resolution on subsequent US scans) or, finally, to kinking of the vascular anastomosis. Instead, a haemodypiù temibili per l'elevata mortalità e la frequente necessità di ritrapianto immediato, è un fattore determinante per la prognosi [24][25][26][27][28]. I segni clinici e laboratoristici, infatti, oltre a essere del tutto aspecifici, insorgono tardivamente, quando ormai i danni al fegato trapiantato sono irreversibili.…”