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EATING DISORDERS (EDs) are compelling health issues focusing on eating and body, characterized by psychopathology and generally accompanied by other psychiatric and medical disorders (Fairburn et al. 2000, Herzog & Eddy 2007). DSM-5 classifies feeding and eating disorders as Pica, rumination disorder, avoidant/restrictive food intake disorder, anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), other specified or unspecified feeding or eating disorder (APA 2013). This study will focus on researches of AN, BN and BED subcategories of EDs from a transdiagnostic framework. Common psychopathology under all these subcategories is observed as struggling with eating and body. However, diagnosis differentiates depending on the strategies developed to control eating and the body as well as the severity of the pathology. This study will initially mention epidemiology and etiology of eating disorders and then will deliver emerging individual and group psychotherapy models. Epidemiology and Etiology in Eating Disorders EDs typically begin in puberty and mainly observed in women. Other psychiatric and medical problems frequently accompany the disorder. Therefore, clinical presentation is complicated. The mindset of EDs cases is overly occupied with body shape and weight. Self-esteem is considered in this respect and various behavior patterns (i.e.: excessive diet, purging, excessive exercise, excessive using of laxative/diuretic, binge eating, etc.) to control body shape and weight are developed. As a result, psychosocial functionality is diminished in most of the cases (Fairburn & Cooper 1989). Prevalence of lifelong EDs are between 1% and 5%; diagnostically this rate is <1-4% in AN, <1-2% in BN and <1-4% in BED in the research by Keski-Rahkonen and Mustelin (2016) throughout Europe in accordance with DSM-5 criteria while subthreshold EDs symptoms are between <1-4%. Eating disorder rate is reported to be increasing each year for Asian countries (Pike & Dunne 2015). Although there is no extensive research in our country in recent years, the rate is between 1-3% like other countries (Vardar & Erzengin 2011). Many factors play a role in etiology of EDs. Biological explanations point out genetic predisposition and neurotransmitter dysregulations (Gordon et al. 2005). It has also been reported that AN and BN differ genetically in the recent years (Hinney & Volckmar 2013). On the other hand, psychodynamic explanations stress on weakness in ego strength stemming from early conflicts, problems in parental relations and denial of femininity, however, studies are observed to be more specific to AN (Zerbe 2001). Cognitive behavioral explanations assume that dysfunctional thoughts related to weight, body, and shape are influential on the development and maintenance of eating disorder symptoms. They also reinforce the disorder through weight control or compensation methods. (Leung et al. 1999, Fairburn 2008). enhanced cognitive-behavioral approach, with its more contemporary view, handles EDs from a transdiagnostic ...
EATING DISORDERS (EDs) are compelling health issues focusing on eating and body, characterized by psychopathology and generally accompanied by other psychiatric and medical disorders (Fairburn et al. 2000, Herzog & Eddy 2007). DSM-5 classifies feeding and eating disorders as Pica, rumination disorder, avoidant/restrictive food intake disorder, anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), other specified or unspecified feeding or eating disorder (APA 2013). This study will focus on researches of AN, BN and BED subcategories of EDs from a transdiagnostic framework. Common psychopathology under all these subcategories is observed as struggling with eating and body. However, diagnosis differentiates depending on the strategies developed to control eating and the body as well as the severity of the pathology. This study will initially mention epidemiology and etiology of eating disorders and then will deliver emerging individual and group psychotherapy models. Epidemiology and Etiology in Eating Disorders EDs typically begin in puberty and mainly observed in women. Other psychiatric and medical problems frequently accompany the disorder. Therefore, clinical presentation is complicated. The mindset of EDs cases is overly occupied with body shape and weight. Self-esteem is considered in this respect and various behavior patterns (i.e.: excessive diet, purging, excessive exercise, excessive using of laxative/diuretic, binge eating, etc.) to control body shape and weight are developed. As a result, psychosocial functionality is diminished in most of the cases (Fairburn & Cooper 1989). Prevalence of lifelong EDs are between 1% and 5%; diagnostically this rate is <1-4% in AN, <1-2% in BN and <1-4% in BED in the research by Keski-Rahkonen and Mustelin (2016) throughout Europe in accordance with DSM-5 criteria while subthreshold EDs symptoms are between <1-4%. Eating disorder rate is reported to be increasing each year for Asian countries (Pike & Dunne 2015). Although there is no extensive research in our country in recent years, the rate is between 1-3% like other countries (Vardar & Erzengin 2011). Many factors play a role in etiology of EDs. Biological explanations point out genetic predisposition and neurotransmitter dysregulations (Gordon et al. 2005). It has also been reported that AN and BN differ genetically in the recent years (Hinney & Volckmar 2013). On the other hand, psychodynamic explanations stress on weakness in ego strength stemming from early conflicts, problems in parental relations and denial of femininity, however, studies are observed to be more specific to AN (Zerbe 2001). Cognitive behavioral explanations assume that dysfunctional thoughts related to weight, body, and shape are influential on the development and maintenance of eating disorder symptoms. They also reinforce the disorder through weight control or compensation methods. (Leung et al. 1999, Fairburn 2008). enhanced cognitive-behavioral approach, with its more contemporary view, handles EDs from a transdiagnostic ...
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