Commotio cordis and ventricular pseudoaneurysm

Fox, Scott; Templeton, Christina; Hancock-Friesen, Camille; Chen, Robert

doi:10.1016/s0828-282x(09)70075-3

Cited by 7 publications

(6 citation statements)

References 4 publications

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“…The leaking site could have been sealed by a thin layer of pericardium and organized thrombus, and later presented with giant LV pseudoaneurysm. Giant LV pseudoaneurysm has been reported to cause mitral regurgitation and compression of adjacent vascular structures [4][5][6] ; however, there was no such complications in the index case. Both echocardiography and CT angiogram are good noninvasive imaging modalities for the diagnosis of pseudoaneurysm [1,3,[6][7][8] .…”

Section: Discussionmentioning

confidence: 84%

Post-myocardial infarction giant left ventricular pseudoaneurysm presenting with severe heart failure

Vijayvergiya

Kumar²,

Rana³

et al. 2012

WJC

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Left ventricle (LV) pseudoaneurysm is a late mechanical complication of myocardial infarction. A giant LV pseudoaneurysm is a rare presentation. We report a case of giant LV pseudoaneurysm in a post-MI patient who presented with gross congestive heart failure. The patient had a successful surgical repair of the aneurysm and had a favorable 3-mo outcome. The imaging modality and surgical treatment of the pseudoaneurysm are discussed.

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Section: Discussionmentioning

confidence: 84%

Post-myocardial infarction giant left ventricular pseudoaneurysm presenting with severe heart failure

Vijayvergiya

Kumar²,

Rana³

et al. 2012

WJC

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“…[ 5 6 ] Victims typically collapse upon impact; 20% of patients remain conscious for several seconds and very few present minutes or hours after impact. [ 7 8 9 ]…”

Section: Discussionmentioning

confidence: 99%

An unusual case of commotio cordis resulting in ventricular flutter

Davey

Quintana

Upadhyay

2018

J Emerg Trauma Shock

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“…Additional investigations of the characteristics of mechanical stretch in commotio cordis indicate that Ca +2 waves are initiated by damaged myocardial tissue, whereby nonuniform muscles are stretched during contraction triggering VF [ 9 , 10 ]. Furthermore, several additional individual variables have been noted to increase VF incidence in multiple animal studies and patient case reports [ 11 , 12 ]. In general, variations of wider QRS and longer QT periods at baseline (possibly influenced by genetic differences in genotypes of repolarization channels) have higher susceptibility to VF.…”

Section: Review Of Ventricular Arrhythmia Mechanisms In Blunt Chesmentioning

confidence: 99%

Mechanisms and Clinical Management of Ventricular Arrhythmias following Blunt Chest Trauma

Wolbrom

Rahman

Tschabrunn

2016

Cardiology Research and Practice

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Nonpenetrating, blunt chest trauma is a serious medical condition with varied clinical presentations and implications. This can be the result of a dense projectile during competitive and recreational sports but may also include other etiologies such as motor vehicle accidents or traumatic falls. In this setting, the manifestation of ventricular arrhythmias has been observed both acutely and chronically. This is based on two entirely separate mechanisms and etiologies requiring different treatments. Ventricular fibrillation can occur immediately after chest wall injury (commotio cordis) and requires rapid defibrillation. Monomorphic ventricular tachycardia can develop in the chronic stage due to underlying structural heart disease long after blunt chest injury. The associated arrhythmogenic tissue may be complex and provides the necessary substrate to form a reentrant VT circuit. Ventricular tachycardia in the absence of overt structural heart disease appears to be focal in nature with rapid termination during ablation. Regardless of the VT mechanism, patients with recurrent episodes, despite antiarrhythmic medication in the chronic stage following blunt chest injury, are likely to require ablation to achieve VT control. This review article will describe the mechanisms, pathophysiology, and treatment of ventricular arrhythmias that occur in both the acute and chronic stages following blunt chest trauma.

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Commotio cordis and ventricular pseudoaneurysm

Cited by 7 publications

References 4 publications

Post-myocardial infarction giant left ventricular pseudoaneurysm presenting with severe heart failure

Post-myocardial infarction giant left ventricular pseudoaneurysm presenting with severe heart failure

An unusual case of commotio cordis resulting in ventricular flutter

Mechanisms and Clinical Management of Ventricular Arrhythmias following Blunt Chest Trauma

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