Comparative Study of Growth of Chinese Infants: Hong Kong versus Guangzhou

Leung, S. S. F.; Peng, Cong; Xu, Yiqun; Liu, K. M.; Quan, Xing; Lui, SL; Davies, D P

doi:10.1093/tropej/40.3.166

Cited by 5 publications

(3 citation statements)

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“…Hong Kong infants were both lighter and shorter than Australian infants (Leung and Davies, 1994). A strong correlation was also observed between the height of Hong Kong parents and their offspnng .…”

Section: Growth Of Chinese Infants -Hong Kongmentioning

confidence: 71%

See 1 more Smart Citation

The growth and feeding patterns of 9 to 12 month old Chinese Canadian infants

Sit

Yeung

et al. 2001

Nutrition Research

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Section: Growth Of Chinese Infants -Hong Kongmentioning

confidence: 71%

“…Solid foods were introduced betwwn four to six rnonüis of age (Leung and Davies, 1994). By eight mnths, the main oalids were congee or soft rice together with steamed fish, meat and vegetables.…”

Section: Lnfant Feeding Practices In Hong Kongmentioning

confidence: 99%

The growth and feeding patterns of 9 to 12 month old Chinese Canadian infants

Sit

Yeung

et al. 2001

Nutrition Research

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“…However, it is also possible that these observations are due to infections that preclude optimal infant growth (32,33) through growth faltering (34). To clarify this question, we took advantage of data on a large population-representative birth cohort from a developed setting, Hong Kong's Children of 1997, in which infections would be expected to have a limited influence on infant growth (35). Specifically, we tested the hypothesis that infectious morbidity in infancy, but not childhood, would be associated with later puberty without affecting growth.…”

mentioning

confidence: 99%

Early Life Infections and Onset of Puberty: Evidence From Hong Kong's Children of 1997 Birth Cohort

Kwok¹,

Leung²,

Lam³

et al. 2011

American Journal of Epidemiology

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As economic development increases, puberty occurs at younger ages, and this could contribute to an increase in the incidence of cardiovascular diseases and hormone-related cancers. The factors that determine pubertal timing are poorly understood. The growth axis that is active during puberty is active in the first 6 months of life and interacts with the immune system. The authors examined whether prior infections, proxied by number of hospital admissions for infections at different ages, were associated with age at pubertal onset (Tanner stage II) using interval-censored regression in the Children of 1997 cohort, which is a population-representative Chinese birth cohort (n = 7,527). Mediation by growth was also examined. Girls, but not boys, who were hospitalized for infections at least twice in the first 6 months of life experienced pubertal onset about 8 months later (mean = 10.3 years, time ratio = 1.08, 95% confidence interval: 1.04, 1.12) than did those without such hospitalizations (mean = 9.6 years) after adjustment for infant characteristics and socioeconomic position (sex interaction: P = 0.02). There were no such associations for infections at 6 months to ≤8 years of age. Growth did not mediate the association. Early infectious morbidity in girls may be associated with later puberty, perhaps via suppression of the gonadotropic axis. The lowering of the number of infections in early life that accompanies economic development could be an additional factor that contributes to earlier puberty.

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