Comparison of Two‐ and Three‐Dimensional Transthoracic Echocardiography in the Assessment of Trabeculations and Trabecular Mass in Left Ventricular Noncompaction

Rajdev, Sanjay; Singh, Anurag; Nanda, F.I.S.C.U. Navin C.; Baysan, Oben; Hsiung, Ming C.

doi:10.1111/j.1540-8175.2007.00504.x

Cited by 25 publications

(27 citation statements)

References 14 publications

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“…32,33 Rarer Metastatic Tumors: Chordomas and melanomas are relatively rare rumors, but melanoma is well known to have a unique tendency to localize in the heart or pericardium. 22,36 The additional benefits derived from using 3D in addition to 2D imaging to assess for noncompaction syndrome were also described by Rajdev et al 37 In their substantially sized review of 21 patients with the left ventricular noncompaction syndrome, 2DTTE underestimated left ventricular trabecular mass and total number of trabeculations compared to RT3DTTE. However, melanoma metastatic to the RV and chordoma metastatic to the right atrium have been identified by RT3DTEE (Fig.…”

Section: Carcinoidmentioning

confidence: 89%

Real Time Three‐Dimensional Echocardiography Evaluation of Intracardiac Masses

2012

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The evaluation of cardiac masses is often a challenge for cardiac imaging techniques. The traditional standby has been two-dimensional echocardiography (2DE). Real time three-dimensional echocardiography (RT3DE) offers incremental value for the evaluation of intracardiac masses by providing more accurate assessment of the size and shape of the mass as well as in some cases, composition of the mass. RT3DE aids with the relationship between the mass and adjacent structures. Therefore, here we discuss the subject of RT3DE evaluation of cardiac masses is reviewed and discussed in detail.

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Section: Carcinoidmentioning

confidence: 89%

Real Time Three‐Dimensional Echocardiography Evaluation of Intracardiac Masses

2012

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“…Real-time 3D echocardiogram is a newer technique which facilitates identifications of noncompacted/ trabeculated myocardium [Engberding et al 2010;Rajdev et al 2007;Baker et al 2006].…”

Section: Diagnosismentioning

confidence: 99%

Left ventricular noncompaction cardiomyopathy: updated review

Udeoji

Philip

Morrissey

et al. 2013

Therapeutic Advances in Cardiovascular Disease

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Abstract:The first case of noncompaction was described in 1932 after an autopsy performed on a newborn infant with aortic atresia/coronary-ventricular fistula. Isolated noncompaction cardiomyopathy was first described in 1984. A review on selected/relevant medical literature was conducted using Pubmed from 1984 to 2013 and the pathogenesis, clinical features, and management are discussed. Left ventricular noncompaction (LVNC) is a relatively rare congenital condition that results from arrest of the normal compaction process of the myocardium during fetal development. LVNC shows variability in its genetic pattern, pathophysiologic findings, and clinical presentations. The genetic heterogeneity, phenotypical overlap, and variety in clinical presentation raised the suspicion that LVNC might just be a morphological variant of other cardiomyopathies, but the American Heart Association classifies LVNC as a primary genetic cardiomyopathy. The familiar type is common and follows a X-linked, autosomal-dominant, or mitochondrial-inheritance pattern (in children). LVNC can occur in isolation or coexist with other cardiac and/or systemic anomalies. The clinical presentations are variable ranging from asymptomatic patients to patients who develop ventricular arrhythmias, thromboembolism, heart failure, and sudden cardiac death. Increased awareness over the last 25 years and improvements in technology have increased the identification of this illness and improved the clinical outcome and prognosis. LVNC is commonly diagnosed by echocardiography. Other useful diagnostic techniques for LVNC include cardiac magnetic resonance imaging, computerized tomography, and left ventriculography. Management is symptom based and patients with symptoms have a poorer prognosis. LVNC is a genetically heterogeneous disorder which can be associated with other anomalies. Making the correct diagnosis is important because of the possible associations and the need for long-term management and screening of living relatives.

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“…In a larger series of NCCM patients, noncompacted and compacted LV segments had comparable increased 3D regional volumes and reduced systolic function suggesting that systolic LV dysfunction is not confined to noncompacted LV segments [19]. In a RT3DE study, both LV trabecular mass and the total number of trabeculations in NCCM patients were significantly underestimated by 2-dimensional (2D) echocardiography as compared to RT3DE [20]. Moreover, the extent of noncompacted myocardium could be identified by RT3DE [21].…”

mentioning

confidence: 85%