Compensatory Eating and Behaviors in Response to Alcohol Consumption Scale

Drunkorexia: is it really “just” a university lifestyle choice?

“…"Drunkorexic" behaviours were measured using the Compensatory Eating and Behaviours in Response to Alcohol Consumption Scale [20,23]. The CEBRACS measures Drunkorexia behaviours across four factors (alcohol effects, bulimia, diet and exercise; and restriction) as well as a total score.…”

“…It was developed based upon the qualitative research of Peralta et al (11) and Burke et al [21]. When compared to the three subscales (drive for thinness, bulimia and body dissatisfaction) of the EDI-2 [22], the CEBRACs was found to be a valid measure [20,23] which is now commonly used in drunkorexia studies [24,25]. The CEBRACS scale is split into three time periods for participants to report any changes in their behaviour before drinking while under the effects of alcohol (during drinking) and after the effects of alcohol have worn off (after drinking).…”

“… because I'm so drunk at the time, the last thing I'm going to think about is calories”: Strengthening the argument for Drunkorexia as a food and alcohol disturbance, evidence from a qualitative study

“…While Drunkorexia is not currently a classified disorder, Rahal, Bryant, Darkes, Menzel, and Thompson (2012a, 2012b), developed the Compensatory Eating and Behaviours in Response to Alcohol Consumption scale (CEBRACs), which is frequently used in research to show the extent and prevalence of the behaviours (Bryant et al., 2012; Hill & Lego, 2019). The debate as to whether Drunkorexia is an eating disorder or substance abuse disorder is still ongoing, with evidence suggesting it cannot solely be classified as either (Griffin & Vogt, 2020; Hill & Lego, 2019; Hunt & Forbush, 2016).…”

Associations between alcohol use-related outcomes and psychopathological symptoms: person- and variable-oriented approaches

“…Shared neurobiological correlates (e.g., areas related to reward processes and behavioral control) and psychological and affective characteristics (e.g., both risk behaviors associated with elevated levels of internalizing symptoms, neuroticism, maladaptive emotion regulation, impulsivity as well as similar motivational mechanisms are shared between them) can explain the aforementioned positive correlation (Ferriter & Ray, 2011;Schulte et al, 2016). Alternatively, the concept of food and alcohol disturbance (FAD) suggested that symptoms and characteristics of EDs (e.g., restrictive tendencies, bulimic characteristics) and alcohol use can be associated functionally (Choquette et al, 2018;Rahal et al, 2012). Namely, one might perform compensatory, calorie restrictive behaviors before (e.g., eating less to get drunk), during (e.g., not eating or eating only low-calorie foods to get drunk or to compensate the calories in alcohol) and after (e.g., skipping meals to compensate the calories due to previous alcohol intake) alcohol use in order to enhance the effects of alcohol or to compensate previous or anticipated calorie intake (Rahal et al, 2012).…”

“…Alternatively, the concept of food and alcohol disturbance (FAD) suggested that symptoms and characteristics of EDs (e.g., restrictive tendencies, bulimic characteristics) and alcohol use can be associated functionally (Choquette et al, 2018;Rahal et al, 2012). Namely, one might perform compensatory, calorie restrictive behaviors before (e.g., eating less to get drunk), during (e.g., not eating or eating only low-calorie foods to get drunk or to compensate the calories in alcohol) and after (e.g., skipping meals to compensate the calories due to previous alcohol intake) alcohol use in order to enhance the effects of alcohol or to compensate previous or anticipated calorie intake (Rahal et al, 2012).…”