Composition and Consumption of Dietary Fats in U.S.A., U.K. and Common Market Countries in 1961

Stroink, J. B. A.

doi:10.1159/000175169

Ann Nutr Metab

1967

DOI: 10.1159/000175169

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Composition and Consumption of Dietary Fats in U.S.A., U.K. and Common Market Countries in 1961

J. B. A. Stroink¹

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“…It might even be better to make a classification according to the amount of calories in the fat components. Table II shows such a classification for the principal fatty foods in the average diet of some countries (Stroink, 1967). The values relate to examples from developed countries, but in other countries such as India, ghee (buffalo butter) and its vegetable substitute, vanaspati, may appreciably contribute to the fat intake.…”

mentioning

confidence: 99%

Synthetic flavors for fatty foods

Keppler¹

1970

J. Agric. Food Chem.

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mentioning

confidence: 99%

Synthetic flavors for fatty foods

Keppler¹

1970

J. Agric. Food Chem.

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Juvenile familial hypertriglyceridemia and growth retardation

Sternowsky¹,

Gaertner

Stahnke³

et al. 1977

Eur J Pediatr

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Familial hypertriglyceridemia or hyperlipoproteinemia type I was detected in three siblings aged 6, 11, and 14 of an otherwise normal Turkish family of 10 members. Initial values ranged from 1780 to 3750 mg/100 ml triglycerides in the milky white serum; cholesterol was normal. Lipoprotein pattern on agarose and acrylamide gel revealed a heavy band of chylomicrons and missing HDL; post-heparin lipolytic activity was decreased to about 30% of normal. Chylomicronemia could be induced by a fat-rich (50% of total calories) diet, but not by carbohydrates. On a low fat diet (5%) during hospitalization chylomicrons disappeared, and triglycerides decreased to about 450 mg/100 ml. Phenocopies of hypertriglyceridemia could be excluded. All three patients were the only members of the family who were small, below the third percentile. Their bone age was retarded from 18 to 30 months. There was no indication for an endocrine cause of the growth retardation: four different stimulation tests revealed normal growth hormone response, thyroid and adrenal functions were not impaired; sexual development was normal. Increased glucose assimilation was observed during intravenous and oral glucose load. Peak serum insulin response was above normal during stimulation tests. The possible etiologic role of hypertriglyceridemia in this concomitant growth retardation is discussed.

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Die Phosphatide in Serum und Leber bei Lebercirrhose

Wolfram¹,

Zöllner²

1971

Klin Wochenschr

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Composition and Consumption of Dietary Fats in U.S.A., U.K. and Common Market Countries in 1961

Cited by 3 publications

References 1 publication

Synthetic flavors for fatty foods

Synthetic flavors for fatty foods

Juvenile familial hypertriglyceridemia and growth retardation

Die Phosphatide in Serum und Leber bei Lebercirrhose

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