Computed axial tomography in the early diagnosis of prostatic abscess

Chia, John K. S.; Longfield, Robert N.; Cook, David H.; Flax, B.L.

doi:10.1016/0002-9343(86)90375-x

Cited by 11 publications

(9 citation statements)

References 6 publications

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“…Worsening suprapubic and perineal painStraddle injury to urethra, urethroplasty, hepatitis CNoCope loop catheter placementVancomycinResolution[33]43MRSADysuria, hesitancy, slow stream, night sweats, flushing, and perineal painIntravenous drug abuse, hepatitis CYesTURPVancomycin, nafcillin, TMP-SMXResolution[34]63MRSAUrinary retention, pyrexiaRecent scrotal drainage, T2DMYesPercutaneous, TURPVancomycinUnknown[35]59SA [unknown susceptibility]Difficulty passing urineRecent salivary gland abscess treated with antibioticsNoTransperineal, TURPCiprofloxacin, erythromycinResolution[36]35SA“clinical acute prostatitis”HIV, bilateral renal abscessesNoNo drainage“IV antibiotics”Resolution[37]42MSSADysuria. urethral discharge, perineal painNoNoTransperinealOxytetracyclineResolution[38]43SA [unknown susceptibility]Dysuria, haematuria, pyrexia, purulent dischargeRecent pelvic infection and pelvic surgeryNoTransurethralGentamicin, carbenicillin.Resolution after relapse[9]54MSSAPerineal painMycosis fungoidesNoTransperineal, TURPOxacillin, tobramycinResolution[39]53MRSAUrinary urgency, fatigue, chills, rigors, and feverDiabetes mellitus, ObesityYesNo drainageVancomycin, TMP-SMX, rifampinResolution[40]50MRSA + PVLAbdominal pain, cough, fever, weight loss, malaiseDiabetes mellitusYesCT guided drainageVancomycin, levofloxacin, daptomycinResolution[41]46MRSAAcute urinary retention, dysuria, constipationInstrumentationYesTransurethralIV Ant...…”

Section: Resultsmentioning

confidence: 99%

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Staphylococcus aureus Prostatic abscess: a clinical case report and a review of the literature

et al. 2017

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BackgroundProstatic abscess is a rare complication of acute bacterial prostatitis and is most commonly caused by Enterobacteriaceae. We report on a case of prostatic abscess caused by Staphylococcus aureus and conduct a review of the literature.Case presentationWe present a case of S. aureus prostatic abscess that was successfully treated with a combination of antibiotic and surgical therapy. The isolate was non–multidrug-resistant, methicillin-resistant Staphylococcus aureus and was genotyped as clonal complex 5, an emerging regional clone that is trimethoprim resistant and Panton-Valentine leukocidin positive. This current case report is the first to describe the use of clindamycin step-down therapy. A literature review identified a further 39 cases of S. aureus prostatic abscesses, of which 26 were methicillin resistant.Conclusions S. aureus is an uncommon cause of prostatic abscess. Optimal management includes both antibiotic therapy and surgical drainage. Our use of clindamycin as step-down therapy was guided by its excellent prostatic penetration.Electronic supplementary materialThe online version of this article (doi:10.1186/s12879-017-2605-4) contains supplementary material, which is available to authorized users.

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Section: Resultsmentioning

confidence: 99%

“…In addition one patient [8] had urinary cultures positive for Candida albicans and one patient [9] was co-infected with E. coli , Bacteroides and Klebsiella pneumoniae .…”

Section: Resultsmentioning

confidence: 99%

Staphylococcus aureus Prostatic abscess: a clinical case report and a review of the literature

et al. 2017

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“…Systemic mycosis caused by Cryptococcus neoformans, Blastomyces dermatitidis, Coccidioides immitis, or Histoplasma capsulatum can involve the prostate gland and cause PA (Bergner et al 1981;Marans et al 1991;Vahlensieck 1987). This is promoted especially in older patients by bladder outlet obstruction caused, for example, by neurogenic bladder, urolithiasis, carcinoma of the prostate and benign prostatic hyperplasia, urinary tract infection, adnexitis, or transurethral instrumentation including previous prostatectomy, prostate needle biopsy, or indwelling catheter (Bartlett et al 1986;Bergner et al 1981;Brawer and Stamey 1987;Chia et al 1986;Cytron et al 1988;Dennis and Donohue 1985;Kadmon et al 1986;Learmonth and Philp 1988;Lentino et al 1984;Marans et al 1991;Mariani et al 1983;Mitchell and Blake 1972;Morrison et a1.1988;Pai and Bhat 1972;Papanicolaou et al 1987;Rorvik and Daehlin 1989;Sohlberg et al 1991;Steinhardt 1988;Sugao et al 1986;Trapnell and Roberts 1970;Vaccaro et al 1986;Trapnell and Roberts 1970;Vaccaro et al 1986;Washecka and Rumancik 1985;Weinberg et al 1985;Woo et al 1987). The route of infection in most cases is canalicular ascending with influx of infected urine into the prostatic ducts (Brawer and Stamey 1987;Meares 1980Trapnell and Roberts 1970).…”

Section: Etiology and Pathogenesismentioning

confidence: 99%

“…Candida albicans may infect the prostate gland by ascending hematogenous route and rarely cause PA (Lentino et al 1984;Vahlensieck 1987). From: Bartlett et al (1978), Bergner et al (1981), Brawer and Stamey (1987), Chia et al (1986), Cytron et al (1988), Dennis and Donohue (1985), , Kadmon et al (1986), Learmonth and Philp (1988), Lentino et al (1984), Marans et al (1991), Mariani et al (1983), Mitchell and Blake (1972), Morrison et al (1988), Pai and Bhat (1972), Papanicolaou et al (1987), Rorvik and Daehlin (1989), Sohlberg et al (1991), Steinhardt (1988), Sugao et al (1986), Trapnell and Roberts (1970), Vaccaro et al (1986), Washecka and Rumancik (1985), Weinberg et al (1985), Woo et al (1987) 50% of reported PA cases are a result of instrumentation or indwelling catheters (Meares 1980. This is promoted especially in older patients by bladder outlet obstruction caused, for example, by neurogenic bladder, urolithiasis, carcinoma of the prostate and benign prostatic hyperplasia, urinary tract infection, adnexitis, or transurethral instrumentation including previous prostatectomy, prostate needle biopsy, or indwelling catheter (Bartlett et al 1986;Bergner et al 1981;Brawer and Stamey 1987;Chia et al 1986;Cytron et al 1988;Dennis and Donohue 1985;Kadmon et al 1986;Learmonth and Philp 1988;…”

Section: Etiology and Pathogenesismentioning

confidence: 99%

“…Less commonly, confluence of micro abscesses is observed, forming PA (Petersen 1986). The dense capsule of the prostate may contain the abscess and favor spontaneous transurethral drainage (5% of cases) (Bartlett et al 1986;Bergner et al 1981;Brawer and Stamey 1987;Chia et al 1986;Cytron et al 1988;Dennis and Donohue 1985;Kadmon et aI. Epithelial cell groups are small and exhibit unclear cell borders (Leistenschneider and Nagel 1978).…”

Section: Pathologymentioning

confidence: 99%

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Prostatitis

1994

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Sol'!cover reprint of the hardcover 1 st edition 1994The use of general descriptive names, registered names, trademarks, etc. in this publication does not imply, even in the absence of a specific statement, that such names are exempt from the relevant protective laws and regulations and therefore free for general use.Typesetting: VII PrefaceProstatitis syndromes continue to be a major problem in urology. About every second man will experience symptoms of prostatitis during his lifetime. A study by the U.S. National Center for Health Statistics showed that approximately 25% of the men seen for urogenital problems suffer from prostatitis.What is the intention of this book? Although investigations in recent years, including microbiologic and immunologic ones, have focused on the correct diagnosis of prostatitis, much confusion still remains regarding its correct classification, its etiology, and the evaluation of diagnostic results. Many clinicians, therefore, tend to lump together several diseases of variable type and sequela as prostatitis.The term "prostatitis" implies an inflammatory disease, but true bacterial infection is detected in only 5 %-1 0% of patients. In fact, most patients reveal no significant bacterial counts in the prostatic fluid. Nonetheless, 40%-60% of these patients suffer from non bacterial prostatitis, as shown by the fact that the prostatic fluid contains elevated leukocyte counts while common uropathogens are not detected.Essentially, three clinical types of real inflammatory prostatitis are widely accepted: acute and chronic bacterial and nonbacterial prostatitis. There remains a condition denoted as prostatodynia, covering patients in whom signs of neither inflammation nor infection are found. Multiple extraprostatic disorders are suspected causes of these complaints, e.g., bladder outlet disorders, detrusor hyperreflexia, pelvic floor tension myalgia, interstitial cystitis, urolithiasis, ostitis pubis, and anogenital disorders. Proven data concerning these special problems are rare, and their association to prostatodynia is unclear. The intention of this book is to provide the reader with a clarification of the etiopathogenesis of prostatitis and the competence to exclude the patient with complaints and symptoms of prostatitis but without true prostatic inflammation, i.e., to identify the man suffering from prostatodynia, thus preventing multiple and useless antibiotic trials. Instead, if the diagnosis prostatodynia is clarified, there may be an indication for, among other things, psychodynamic investigation and therapy.This book concentrates on clinical prostatitis strictu sensu and does not treat the various types of histologically detected inflammation of the prostate, e.g., "prostatitis" associated with benign prostatic hypertrophy. Very rarely, prostatectomy specimens of younger patients or autopsy samples of prostates x Preface without evidence of hyperplasia demonstrate inflammatory infiltrates. In men up to the age of 40, prostatitis is mainly located in the peripheral gland, no...

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