Down syndrome (DS) is among the intellectual disabilities most commonly associated with genetic factors (e.g., Lanfranchi et al., 2012). In the nonspecific intellectual disability (NSID) group, identifying the causes of intellectual disability (ID) during diagnosis has proven unsuccessful. Individuals with DS, when compared with peers of a similar mental age, show alterations in different components related to memory abilities (Godfrey & Lee, 2018), specifically, impaired verbal short-term memory (STM) (Lanfranchi et al., 2004), relatively preserved visuospatial STM (Carretti et al., 2013); and impairment in both verbal and visuospatial working memory (Lanfranchi et al., 2012). In terms of executive functioning, Lanfranchi et al. (2010) and Loveall et al. (2017) found impairment effects among adolescents with DS, although this functioning may be better retained at preschool age (Loveall et al., 2017). Cognitive abilities of individuals with NSID are generally considered delayed, especially when the comparison group presents similar nonverbal development (Lifshitz et al., 2016). Carretti et al. (2010) point to limitations in tasks that impose a high cognitive load (e.g., dual and updating word span tasks), with verbal and visuospatial functioning in STM, and tasks with a "moderate" cognitive load (e.g., backward and selective word span tasks) consistent with their cognitive development. Other studies offer a more unfavorable profile of NSID. Schuchardt et al. (2011) identified deficits in verbal storage capacity yet visuospatial processing capacity consistent with