Congenital Absence of the Right Pulmonary Artery

Madoff, Irving M.; Gaensler, Edward A.; Strieder, John W.

doi:10.1056/nejm195207312470501

Cited by 89 publications

(8 citation statements)

References 18 publications

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“…Efforts have been made to measure the volume rate of flow through such vessels (13)(14)(15)(16)(17). These physiologic studies have been predominantly oriented toward the assessment of the hemodynamic burden which is imposed upon the left heart by the systemic contribution to pulmonary blood flow.…”

mentioning

confidence: 99%

The “Effective” Pulmonary Collateral Blood Flow in Man1

Fishman¹,

Turino²,

Brandfonbrener³

et al. 1958

J. Clin. Invest.

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confidence: 99%

The “Effective” Pulmonary Collateral Blood Flow in Man1

Fishman¹,

Turino²,

Brandfonbrener³

et al. 1958

J. Clin. Invest.

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“…Most of the collateral vessels hypertrophy following birth, and produce numerous vascular channels in the submucosa of the bronchial walls [4]. Bronchial blood flow to the affected lung may be increased 15-25 fold and can account for up to one third of the left ventricular output [1,6]. The incidence of pulmonary hypertension in the isolated cases is 19% [1].…”

Section: Discussionmentioning

confidence: 99%

A patient with haemoptysis and a smaller right lung

Kuo¹,

Wu²,

Lee³

1996

Eur Respir J

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A A p pa at ti ie en nt t w wi it th h h ha ae em mo op pt ty ys si is s a an nd d a a s sm ma al ll le er r r ri ig gh ht t l lu un ng g Case historyA 41-year-old businessman was admitted to National Taiwan University Hospital for evaluation of a 2-week history of haemoptysis productive of 50-100 mL of fresh blood daily. The patient stated that haemoptysis had first occurred 10 yrs earlier, had lasted for about 1 month and had spontaneously subsided. Both episodes of haemoptysis were preceded by flu-like symptoms. There was no past history of epistaxis, exertional dyspnoea, or loss of body weight.Physical examination revealed a well-developed man in no distress. His blood pressure was 112/70 mmHg, pulse 96 beats . min -l and respiratory rate 22 breaths . min -1 . There was a slightly decreased chest wall excursion, and breath sounds in the right hemithorax. No audible bruits were heard over the chest. Heart sounds were regular, without murmurs. No clubbing, cyanosis, telangiectasia or oedema were noted. Arterial blood gases in room air showed: a pH of 7.41; arterial carbon dioxide tension (Pa,CO 2 ) of 4.9 kPa (36.5 mmHg); and arterial oxygen tension (Pa,O 2 ) of 13.1 kPa (98.3 mmHg). The sputum was negative for acid-fast bacilli, fungi and malignant cells. An electrocardiogram was normal. Echocardiography showed normal cardiac size and good left ventricular contractility, with no evidence of congenital cardiac lesions or pulmonary hypertension. On bronchoscopy, no endobronchial lesion or definite site of bleeding was found.Imaging studies, including a posteroanterior (PA) chest radiograph ( fig. l), and computed tomography (CT) scan at the level of the aorticopulmonary window ( fig. 2) are shown.

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“…Such a decision may also be reinforced by the failure of bronchospirometry to demonstrate a significant oxygen consumption by the affected lung. Oxygen uptakes varying from 0 to 7% of the total consumption have been found under such circumstances (Madoff et al, 1952;Smart and Pattinson, 1956;Fishman, Turino, Brandfonbrener, and Himmelstein, 1958;Tabakin, Hanson, Adhikari, and Miller, 1960), suggesting little, if any, participation in gas exchange by the abnormal lung.…”

mentioning

confidence: 99%

“…Congenital absence of a main pulmonary artery to one lung is a well-documented anomaly (Fraentzel, 1868;Miller, 1937;Blalock, 1948;Findlay and Maier, 1951;Madoff, Gaensler, and Strieder, 1952) and its association with unilateral cystic lung disease was emphasized in particular by Mannix and Haight (1955) and by Steinberg (1958). According to Madoff and others, a preponderance of congenital cardiac defects is apparent in patients with an absence of the left pulmonary artery.…”

mentioning

confidence: 99%