Brain Edema XI 2000
DOI: 10.1007/978-3-7091-6346-7_96
|View full text |Cite
|
Sign up to set email alerts
|

Continuous Monitoring of ICP and CPP Following ICH and its Relationship to Clinical, Radiological and Surgical Parameters

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
2
1

Citation Types

0
31
1

Year Published

2008
2008
2024
2024

Publication Types

Select...
7
2

Relationship

0
9

Authors

Journals

citations
Cited by 42 publications
(32 citation statements)
references
References 7 publications
0
31
1
Order By: Relevance
“…5 After the acute phase, high morbidity and mortality are essentially caused by the evolution of a peri-hemorrhagic, space-occupying edema associated with gradually increasing intracranial pressure (ICP). 6,7 Although the natural course of edema formation is still not fully understood, edema commonly increases strongly during the first week and reaches its maximum during the second week after bleeding onset. 8 To date, there is no effective therapy for spontaneous ICH.…”
mentioning
confidence: 99%
“…5 After the acute phase, high morbidity and mortality are essentially caused by the evolution of a peri-hemorrhagic, space-occupying edema associated with gradually increasing intracranial pressure (ICP). 6,7 Although the natural course of edema formation is still not fully understood, edema commonly increases strongly during the first week and reaches its maximum during the second week after bleeding onset. 8 To date, there is no effective therapy for spontaneous ICH.…”
mentioning
confidence: 99%
“…3 After the acute phase, neurological deterioration is mainly attributed to the evolution of perihemorrhagic edema with mass effect and increasing intracranial pressure (ICP). 4 Although only a few studies have investigated the course of edema evolution, it seems that there is a strong increase during the first week and a peak in the second week after initial bleeding. 5 To date, no medical or surgical therapy for ICH and perihemorrhagic edema has been shown to improve outcome after ICH.…”
mentioning
confidence: 99%
“…Mayer et al investigated the potential use of rFVIIa for improving outcome and established that this therapy may in fact improve hematoma volume, but its impact on outcomes was limited (18). Hematoma volume & growth does correlate with various outcome measures (19)(20)(21), but so does perihemorrhagic edema (22)(23)(24)(25). ICH is associated with secondary injury characteristics that are similar to ischemia and ischemia-reperfusion, including blood-brain barrier disruption, inflammation and edema.…”
Section: Therapeutic Hypothermia For Intracerebral Hemmorhagementioning
confidence: 99%
“…The edema progresses through three phases related initially to hydrostatic forces & clot retraction, then activation of the coagulation cascade and thrombin formation and later, via RBC lysis and hemoglobin-induced neuronal toxicity (26). This edema -termed perihemorrhagic edema -has been associated with poor outcomes (22,23,25). Data from animal models of ICH suggest that hypothermia can improve these injurious processes, but not outcomes (27)(28)(29)(30)(31)(32)(33).…”
Section: Therapeutic Hypothermia For Intracerebral Hemmorhagementioning
confidence: 99%