Contribution of Peer Deviancy Training to the Early Development of Conduct Problems: Mediators and Moderators

Snyder, James J.; McEachern, Amber D.; Schrepferman, Lynn M.; Just, Christy L.; Jenkins, Melissa; Roberts, Shani Roshelle; Lofgreen, Ashton M.

doi:10.1016/j.beth.2009.05.001

Cited by 40 publications

(49 citation statements)

References 30 publications

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“…One possible explanation for differences in findings between sibling-comparison and the biometric model is that childhood CPs are a direct manifestation of the same temperamental extreme as fussiness, consistent with the spectrum model of temperament-psychopathology association (Clark et al 1994) and passive gene-environment correlation. Another possibility is that ASB is associated with fussy temperament as a result of intervening environmental exposures that are evoked (e.g., coercive parent-child interaction; Patterson, Reid, and Dishion 1992) or selected (e.g., deviant peer groups; Snyder et al 2010) by children with fussy temperaments, consistent with evocative and active gene-environment correlations.…”

Section: Discussionmentioning

confidence: 94%

Genetic and Environmental Contributions to Associations between Infant Fussy Temperament and Antisocial Behavior in Childhood and Adolescence

et al. 2016

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Previous research suggests that fussy temperament in infancy predicts risk for later antisocial behavior (ASB) in childhood and adolescence. It remains unclear, however, to what extent infant fussiness is related to later ASB through causal processes or if they both reflect the same family risk factors for ASB. The current study used two approaches, the comparison of siblings and bivariate biometric modeling, to reduce familial confounding and examine genetic and environmental influences on associations between fussiness in the first two years of life and ASB in childhood and late adolescence. Analyses were conducted on data from a prospective cohort (9,237 at 4-9 years and 7,034 at 14-17years) who are the offspring of a nationally representative sample of U.S. women. In the full sample, fussiness predicted both child and adolescent ASB to small but significant extents, controlling for a wide range of measured child and family-level covariates. When siblings who differed in their fussiness were compared, fussiness predicted ASB in childhood, but not ASB during adolescence. Furthermore, results from a bivariate Cholesky model suggested that even the association of fussiness with childhood ASB found when comparing siblings is attributable to familial factors. That is, although families with infants who are higher in fussiness also tend to have children and adolescents who engage in greater ASB, the hypothesis that infant fussiness has an environmentally mediated impact on the development of future ASB was not strongly supported.

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Section: Discussionmentioning

confidence: 94%

Genetic and Environmental Contributions to Associations between Infant Fussy Temperament and Antisocial Behavior in Childhood and Adolescence

et al. 2016

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“…Snyder et al have suggested that such interactional processes serves as cognitive models and verbal instructions for promoting anti-social behaviors in other social settings [32]. Another aspect of deviancy training is that deviant peers educate each other on how to avoid adult surveillance [32].…”

Section: Discussionmentioning

confidence: 99%

“…Dishion et al have shown through the concept of deviancy training that affiliation with others who have similar behavioral problems facilitates discourse about and modeling of anti-social behavior [30]. Such social interactions have been demonstrated to promote the onset and development of conduct problems [30–33], including externalizing symptoms [32,34]. Thus, the more friends a child or adolescent has who are aggressive, the greater this social influence supports aggressive behavior [32] and the more likely it is that those friendships may lead to externalizing behaviors.…”

Section: Introductionmentioning

confidence: 99%

“…Such social interactions have been demonstrated to promote the onset and development of conduct problems [30–33], including externalizing symptoms [32,34]. Thus, the more friends a child or adolescent has who are aggressive, the greater this social influence supports aggressive behavior [32] and the more likely it is that those friendships may lead to externalizing behaviors. [35].…”

Section: Introductionmentioning

confidence: 99%

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Childhood friendships and psychological difficulties in young adulthood: an 18-year follow-up study

Sakyi

Surkan

Fombonne

et al. 2014

Eur Child Adolesc Psychiatry

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Childhood friendships have been shown to impact mental health over the short-term, however, it is unclear whether these effects are sustained into young adulthood. We studied the prospective association between childhood friendships and psychological difficulties in young adulthood. Data come from 1103 French 22–35 year olds participating in the TEMPO study. Childhood friendships were ascertained in 1991 when participants were 4–16 years old. Psychological difficulties were measured in 2009 using the Adult Self-Report. Logistic regression models controlled for participants’ age, sex, childhood psychological difficulties and parental characteristics. Young adults who had no childhood friends had higher odds of psychological difficulties than those with at least one friend: (adjusted ORs: 2.45; 95% CI: 1.32–4.66, p=0.01 for high internalizing symptoms; 1.81; 95% CI: 0.94–3.54, p=0.08 for high externalizing symptoms). Social relations early in life may have consequences for adult psychological well-being.

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“…Later work clarified how peer reinforcement and exposure to deviant talk predict 3-year growth in covert aggression through encouragement of the child's own deviant talk (Snyder et al 2008(Snyder et al , 2010.…”

mentioning

confidence: 99%

Peer-Nominated Deviant Talk Within Residential Treatment: Individual and Group Influences on Treatment Response

Zakriski

Wright

Cardoos

2011

J Abnorm Child Psychol

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This research examined deviant talk during summer residential treatment using peer nominations and extensive field observations. Participants were 239 youth (M (age) = 12.62, SD = 2.60; 67% male), nested in 26 treatment groups. Deviant talk was present in this setting, showed individual differences, and increased over time, especially for younger boys. As expected, its relationship to treatment response was moderated by peer behavior. Initial levels of individual deviant talk were related to clinical improvement, but primarily when peer deviant talk was low. Initial levels of peer deviant talk were related to higher than expected end of treatment aggression, especially for youth who were high in deviant talk. Deviant talk effects were observed for staff impressions of change and observations of aggression and adjustment. Initial antisocial behavior affected whether individual or peer levels of deviant talk more heavily influenced treatment response. Implications for clinical assessment and treatment monitoring are discussed.

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Contribution of Peer Deviancy Training to the Early Development of Conduct Problems: Mediators and Moderators

Cited by 40 publications

References 30 publications

Genetic and Environmental Contributions to Associations between Infant Fussy Temperament and Antisocial Behavior in Childhood and Adolescence

Genetic and Environmental Contributions to Associations between Infant Fussy Temperament and Antisocial Behavior in Childhood and Adolescence

Childhood friendships and psychological difficulties in young adulthood: an 18-year follow-up study

Peer-Nominated Deviant Talk Within Residential Treatment: Individual and Group Influences on Treatment Response

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