Neonatal acidosis, generally defined as an umbilical artery (UA) pH of less than 7.00, 1 has been linked to significant neonatal morbidity, including hypoxic ischemic encephalopathy, intraventricular hemorrhage, cerebral palsy, seizures, and increased rates of neonatal intensive care unit (NICU) admissions and neonatal mortality. [2][3][4][5][6] These associations are thought to be mediated by peripartum hypoxic injury to the fetus, which results in tissue ischemia and increased anaerobic metabolism that is then reflected in a low UA pH at birth.Along with other measures of neonatal well-being, such as APGAR scores, UA pH has been used as a quality indicator among labor wards and as part of the diagnostic and prognostic pathways for neonatal hypoxic-ischemic injury. [1][2][3][4]7,8 However, assessment of pH poses significant limitations if used as a proxy measure for metabolic status. pH is exponentially altered by small changes in respiratory and metabolic function. 9 As such,