Coronary artery bypass grafting using two different anesthetic techniques: Part 3: Adrenergic responses

Liem, Tiong H.; Booij, L.H.D.J.; Gielen, M.J.M.; Hasenbos, M.A.W.M.; Egmond, J. van

doi:10.1016/1053-0770(92)90191-9

Cited by 87 publications

(27 citation statements)

References 28 publications

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“…The ability of TEA to block the stress hormone response to upper abdominal and cardiac surgery is very variable, particularly with respect to cortisol release [11,18,19]. Stress hormones (epinephrine, norepinephrine, cortisol, glucagon, growth hormone) are not maximal during cardiac surgery but rather they continue to increase dramatically some hours afterwards and are still elevated 2 days after surgery [19,20].…”

Section: Discussionmentioning

confidence: 99%

Effects of thoracic epidural analgesia on glucose homeostasis after cardiac surgery in patients with and without diabetes mellitus

Anderson

Ehrenberg

Barr

et al. 2005

European Journal of Anaesthesiology

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Section: Discussionmentioning

confidence: 99%

Effects of thoracic epidural analgesia on glucose homeostasis after cardiac surgery in patients with and without diabetes mellitus

Anderson

Ehrenberg

Barr

et al. 2005

European Journal of Anaesthesiology

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“…1991, Kriemanetal. 1992, Liem et al 1992). Regarding the possibility of technical interference, nicotinamide derivatives have not altered results of the HPLC method (Young et al 1972), and benzodiazepines are not known to interfere with catecholamine or with VMA-HVA assays using HPLC methods (Peyrin 1991).…”

Section: Plasma Prolactinmentioning

confidence: 94%

Catecholamines in Autistic Disorder: Effects of Amisulpride and Bromocriptine in a Controlled Crossover Study

Dollfus¹,

Petit²,

Garnier³

et al. 1993

Journal of Child and Adolescent Psychopharmacology

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The biochemical effects on catecholaminergic systems of the dopamine antagonist amisulpride and the dopamine agonist bromocriptine were evaluated in a double-blind, randomized, crossover study in children with autistic disorder. Plasma levels of dopamine, norepinephrine, and epinephrine; urinary concentrations of homovanillic acid (HVA), vanillyImandelic acid (VMA) and 3-methoxy-4-hydroxyphenylethylene glycol (MHPG); and plasma prolactin were measured. At doses of amisulpride and bromocriptine that had the expected opposing effects on plasma prolactin, the drugs' effects on the catecholaminergic systems were similar. Both agents unexpectedly lowered urinary HVA (total, free, and sulfated) although only amisulpride decreased the HVA levels significantly. This paradoxical decrease in HVA levels suggests that both dopamine agonists and antagonists could act on autoreceptors or presynaptic dopaminergic receptors in the central nervous system. There was no significant action of either drug on plasma epinephrine, urinary VMA, or urinary MHPG, suggesting that neither drug altered norepinephrine or epinephrine systems; however, a weak increase of plasma norepinephrine occurred after amisulpride treatment, consistent with effects observed with other neuroleptics. Neither agent altered plasma dopamine, suggesting that peripheral dopamine metabolism was unchanged. The clinical effects of amisulpride and bromocriptine have been reported to be unexpectedly complementary. The complementary clinical effects of a dopamine agonist and dopamine antagonist might speculatively be related to their similar action on dopamine autoreceptors, leading to a correction of the dopaminergic hyperactivity that has been postulated in autistic children.

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“…Furthermore, Liem and coworkers [44] found a reduced increase in norepinephrine and less variability in epinephrine plasma concentrations when general anaesthesia was combined with TEA during CABG. Cortisol release was augmented during the bypass period in the general anaesthesia/TEA group rather than in the general anaesthesia group.…”

Section: Regional Anaesthesia For Coronary Artery Bypass Graftingmentioning

confidence: 98%

Regional anaesthesia in patients at coronary risk for noncardiac and cardiac surgery

Möllhoff

Theilmeier

2001

Current Opinion in Anaesthesiology

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This review presents a brief overview about the role of regional anaesthesia in patients at risk for myocardial ischemia and/or infarction after cardiac and noncardiac surgical procedures. It includes pathophysiological insights in the problems of plaque rupture and the possible interactions by the use of regional anaesthesia. Special emphasis is put on the subject of thoracic epidural anaesthesia with newer studies showing improvement in relief of angina and improvement of global systolic and diastolic function in patients with coronary artery disease.

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Coronary artery bypass grafting using two different anesthetic techniques: Part 3: Adrenergic responses

Cited by 87 publications

References 28 publications

Effects of thoracic epidural analgesia on glucose homeostasis after cardiac surgery in patients with and without diabetes mellitus

Effects of thoracic epidural analgesia on glucose homeostasis after cardiac surgery in patients with and without diabetes mellitus

Catecholamines in Autistic Disorder: Effects of Amisulpride and Bromocriptine in a Controlled Crossover Study

Regional anaesthesia in patients at coronary risk for noncardiac and cardiac surgery

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