Coronary sinus thrombus without spontaneous contrast

Spontaneous Coronary Sinus Thrombosis in a Patient With Pulmonary Embolism and No Prior Cardiac Instrumentation

“… Floria et al. 2016 9 Abdominal pain, nausea, vomiting, and diarrhea 27 years Male Not mentioned Psoas muscle abscess secondary to Crohn’s disease Deceased. Martin et al.…”

“…14 In the case of spontaneous CS thrombosis, the pathogenesis is unclear, with published data showing different underlying pathological processes with a predominance of inflammatory processes. 5 , 8 , 9 , 10 , 11 In asymptomatic patients, CS thrombosis is often detected incidentally by contrast-enhanced CT scan or angiography performed to evaluate other diagnoses. A TTE may reveal a dilated RA with dilated CS.…”

Coronary sinus endocarditis in a hemodialysis patient: A case report and review of literature

“…Similar to CS vegetation, spontaneous CS thrombosis without iatrogenic injury has been reported in less than 15 cases. CS thrombosis mostly occurred in conditions leading to a static status of the right atrium, including atrial fibrillation[ 18 - 21 ], right heart failure with severe pulmonary hypertension[ 18 , 19 , 22 , 23 ], or hypercoagulable conditions such as severe inflammation/infection[ 20 , 24 - 26 ] and malignancy[ 27 ]; this presentation is similar to that of thrombosis at other sites. Patients with CS thrombosis primarily have symptoms and signs associated with partial or complete obstruction of the CS, but no symptoms and signs of infection.…”

“…For example, chest pain, ST-segment elevation[ 28 ], or hemodynamic collapse and subsequent sudden death[ 22 , 24 , 25 , 27 ] have been reported. CS thrombosis with complete obstruction required emergency surgery[ 19 , 26 ]; however, thrombosis without obstructive signs responded to anti-coagulation therapy[ 18 , 20 ].…”

Coronary sinus thrombosis: Echocardiographic visualization in a patient with known risk factors

“…Management of coronary sinus thrombosis is also poorly described, although thrombectomy and subsequent anticoagulation with heparin bridge to vitamin K antagonist therapy has been employed in both hemodynamically stable and unstable patients . Anticoagulation alone with low‐molecular weight heparin as a bridge to coumadin or novel oral anticoagulant therapy has also been utilized in clinically stable patients . Our patient exhibited a renewed interest in adherence to therapy and apixaban was chosen to be continued given the patient's clinical stability and patient preference.…”