Correction: Corrigendum: Intestinal microbiota as a tetrahydrobiopterin exogenous source in hph-1 mice

Belik, Jaques; Shifrin, Yulia; Arning, Erland; Pan, Jingyi; Daigneault, Michelle; Allen‐Vercoe, Emma

doi:10.1038/srep44161

Cited by 3 publications

(1 citation statement)

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“…The conversion of tyrosine into dopamine by tyrosine hydroxylase (TH) is the rate-limiting process of dopamine synthesis, and tetrahydrobiopterin acts as a coenzyme in this process [ 137 ]. Dopamine is mainly synthesized in brain neurons, but the metabolic pathway of phenylalanine-tyrosine-dopa-dopamine in microorganisms has also been confirmed [ 138 ]. Recent studies have pointed out that BBR can regulate the phenylalanine-tyrosine-dopa-dopamine metabolic process of the gut microbiota to promote the production of intestinal dopa/dopamine and transfer to the circulatory system, from which it can ultimately reach the brain and improve Parkinson's symptoms.…”

Section: Bbr Directly Regulates Gut Microbiotamentioning

confidence: 99%

Interactions between gut microbiota and berberine, a necessary procedure to understand the mechanisms of berberine

Cheng

Liu

Tan

et al. 2022

Journal of Pharmaceutical Analysis

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Section: Bbr Directly Regulates Gut Microbiotamentioning

confidence: 99%

Interactions between gut microbiota and berberine, a necessary procedure to understand the mechanisms of berberine

Cheng

Liu

Tan

et al. 2022

Journal of Pharmaceutical Analysis

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Animal Models of Congenital Gastrointestinal Maladies

Smith

Francis

Kim

et al. 2020

Advances in Experimental Medicine and Biology

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Dysbiosis of intestinal microbiota in autism spectrum disorders: new horizons in search for pathogenetic approaches to therapy. Part 2. Gut–brain axis in pathogenesis of autism spectrum disorders

Благонравова¹,

Жиляева²,

Квашнина³

2021

Journal of microbiology, epidemiology and immunobiology

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The second part of the literature review on the role of intestinal microbiota disturbances in the pathogenesis of autism spectrum disorders (ASD) is devoted to the analysis of published literature on the possible mechanisms underlying the impact of intestinal dysbiosis on the function of the central nervous system and symptoms of ASD and vice versa, the effect of the nervous system on the intestinal microbiota. The hypotheses of slow inflammation, hyperserotoninemia, the production of toxic metabolites of the intestinal microbiota, impaired intestinal wall permeability, and the effect of intestinal dysbiosis on the synthesis of amino acids, vitamins and other biologically active substances that are potentially involved in the etiology and pathogenesis of ASD are considered. Available to date experimental and clinical data supporting these hypotheses are presented. The main mechanisms of the so-called gut-brain axis, which may be related to the pathogenesis of ASD, are formulated.

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Correction: Corrigendum: Intestinal microbiota as a tetrahydrobiopterin exogenous source in hph-1 mice

Abstract: In the original version of this Article, all instances of "Adlercreutzia equolifaciens" were incorrectly given as "Aldercreutzia equolifaciens". This error has now been corrected in the PDF and HTML versions of the Article.

Cited by 3 publications

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Interactions between gut microbiota and berberine, a necessary procedure to understand the mechanisms of berberine

Interactions between gut microbiota and berberine, a necessary procedure to understand the mechanisms of berberine

Animal Models of Congenital Gastrointestinal Maladies

Dysbiosis of intestinal microbiota in autism spectrum disorders: new horizons in search for pathogenetic approaches to therapy. Part 2. Gut–brain axis in pathogenesis of autism spectrum disorders

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