Correction of postischemic disturbances in the hemispheres of the brain using precursors of succinyl-CoA biosynthesis

Use of CoA Biosynthesis Modulators and Selenoprotein Model Substance in Correction of Brain Ischemic and Reperfusion Injuries

“…Selenium is an essential microelement in different brain functions [119][120][121][122][123][124]. Neuroprotective potential of selenium is realized through the expression of selenoproteins: glutathione peroxidase, thioredoxine reductases, methionine sulfoxide reductases, selenoproteins P and R, which participate in regulation of the oxidation-reduction state of the neurones and glial cells under both physiologic conditions, and during oxidation stress [125,126,128].…”