Corrigendum to: Antiplatelet therapy in patients with conservatively managed spontaneous coronary artery dissection from the multicentre DISCO registry

Cerrato, Enrico; Giacobbe, Federico; Quadri, Giorgio; Macaya, Fernando; Bianco, Matteo; Mori, Ricardo; Biolè, Carlo Alberto; Boi, Alberto; Bettari, Luca; Rolfo, Cristina; Ferrari, Fabio; Annibali, Gianmarco; Scappaticci, Massimiliano; Pavani, Marco; Barbero, Umberto; Buccheri, Dario; Cavallino, Chiara; Lombardi, Primiano; Bernelli, Chiara; D’Ascenzo, Fabrizio; Infantino, Vincenzo; Gambino, Alfonso; Cinconze, Sebastian; Rognoni, Andrea; Montagna, Laura; Porto, Italo; Musumeci, Giuseppe; Escaned, Javier; Varbella, Ferdinando

doi:10.1093/eurheartj/ehab720

Cited by 15 publications

(19 citation statements)

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“…This is in line with the American Heart Association recommendation for DAPT use as in case of non SCAD PCI . 9,10 In conservative managed SCAD cases, though, a recently published European registry found DAPT regime to be associated with increased risk of non fatal myocardial infarct and unplanned coronary revascularization compared to single anti platelet agent, in addition to higher bleeding risk 11 . We also administered intra procedural tirofiban as in the IVUS run it was evident that a large organized hematoma had already formed in the false lumen which will not have been possible to be fenestrated using CB and further expansion of the hematoma could compromise the LAD flow and thus adversely impact on downstream flow to the myocardium and on left ventricular function.…”

Section: Discussionmentioning

confidence: 99%

“…7 found DAPT regime to be associated with increased risk of non fatal myocardial infarct and unplanned coronary revascularization compared to single anti platelet agent, in addition to higher bleeding risk. 11 We also administered intra procedural tirofiban as in the IVUS run it was evident that a large organized hematoma had already formed in the false lumen which will not have been possible to be fenestrated using CB and further expansion of the hematoma could compromise the LAD flow and thus adversely impact on downstream flow to the myocardium and on left ventricular function. At present, there are insufficient data regarding the use of GPIIb/IIIa inhibitors in SCAD 9 and cannot be recommended.…”

Section: Discussionmentioning

confidence: 99%

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Balloon assisted hematoma fenestration in spontaneous coronary artery dissection. Case presentation and literature review

Besis

Biase²,

Subkovas³

et al. 2023

Cathet Cardio Intervent

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The management of spontaneous coronary artery dissection (SCAD) is lacking randomized data. This also holds for SCAD with ST‐segment elevation myocardial infarction where stenting has been used to restore coronary flow. This approach can be associated with many drawbacks. Therefore, we present an alternative approach to stenting when coronary flow cannot be restored using cutting balloons alone.

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Balloon assisted hematoma fenestration in spontaneous coronary artery dissection. Case presentation and literature review

Besis

Biase²,

Subkovas³

et al. 2023

Cathet Cardio Intervent

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“…2,67 Data from the 'DIssezioni Spontanee COronariche' (DISCO) registry suggest that in conservatively managed patients, DAPT may indeed be associated with an increased risk of MACE compared with single antiplatelet therapy, thus indicating that at least DAPT should be avoided in SCAD patients. 83 According to expert consensus, in the context of ACS due to angiographically documented SCAD, anticoagulation therapy should be discontinued. 77,84 Finally, it has been reported that SCAD may be associated with some inflammatory conditions.…”

Section: Spontaneous Coronary Artery Dissectionmentioning

confidence: 99%

“…However, antiplatelet agents present a theoretical increased risk of bleeding and propagation of the dissection 2,67 . Data from the ‘DIssezioni Spontanee COronariche’ (DISCO) registry suggest that in conservatively managed patients, DAPT may indeed be associated with an increased risk of MACE compared with single antiplatelet therapy, thus indicating that at least DAPT should be avoided in SCAD patients 83 …”

Section: Mechanisms and Specific Therapy Of Myocardial Infarction Wit...mentioning

confidence: 99%

Myocardial infarction with nonobstructive coronary arteries: from pathophysiology to therapeutic strategies

Foà

Canton

Bodega

et al. 2023

Journal of Cardiovascular Medicine

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Myocardial infarction with nonobstructive coronary arteries (MINOCA) is a heterogeneous group of clinical entities characterized by clinical evidence of acute myocardial infarction (AMI) with normal or near-normal coronary arteries on coronary angiography (stenosis < 50%) and without an over the alternative diagnosis for the acute presentation. Its prevalence ranges from 6% to 11% among all patients with AMI, with a predominance of young, nonwhite females with fewer traditional risks than those with an obstructive coronary artery disease (MI-CAD). MINOCA can be due to either epicardial causes such as rupture or fissuring of unstable nonobstructive atherosclerotic plaque, coronary artery spasm, spontaneous coronary dissection and cardioembolism in-situ or microvascular causes. Besides, also type-2 AMI due to supply-demand mismatch and Takotsubo syndrome must be considered as a possible MINOCA cause. Because of the complex etiology and a limited amount of evidence, there is still some confusion around the management and treatment of these patients. Therefore, the key focus of this condition is to identify the underlying individual mechanisms to achieve patient-specific treatments. Clinical history, electrocardiogram, echocardiography, and coronary angiography represent the first-level diagnostic investigations, but coronary imaging with intravascular ultrasound and optical coherent tomography, coronary physiology testing, and cardiac magnetic resonance imaging offer additional information to understand the underlying cause of MINOCA. Although the prognosis is slightly better compared with MI-CAD patients, MINOCA is not always benign and depends on the etiopathology. This review analyzes all possible pathophysiological mechanisms that could lead to MINOCA and provides the most specific and appropriate therapeutic approach in each scenario.

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“…Pasien yang dilakukan PCI mengikuti pedoman akan mendapakan dual antiplatelet therapy (DAPT) [30]. Pada penelitian cohort membandingkan single antiplatelet therapy (SAPT) dan DAPT, didapatkan terjadi peningkatan kejadian MACE pada DAPT dibandingkan SAPT [37]. Obat golongan statin diberikan hanya pada hiperglikemia atau ada risiko ateroskerosis [38].…”

Section: Manajemen Akutunclassified

Diseksi Spontan Arteri Koroner: Diagnosis dan Manajemen

Laksono¹,

Subandra²

2023

J. Sains Kes.

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Diseksi spontan arteri koroner (Spontaneous Coronary Artery Dissection: SCAD) salah satu penyebab kasus SKA yang jarang tetapi meningkat insidensi pada wanita muda dengan penilaian risiko kardivaskular rendah. Selain itu SCAD memiliki faktor risiko terhadap kehamilan, dan penyakit fibromuscular dysplasia. Penegakan diagnosis SCAD dengan angiografi koroner menjadi tantangan dikarenakan karakteristik SCAD yang mirip dengan penyebab ACS lainnya dan sering kali terlewat. Diharapkan SCAD dapat menjadi perhatian penelitiaan untuk mendukung penegakan diagnosis dan manajemen guna menghasilkan keluaran lebih baik.

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Corrigendum to: Antiplatelet therapy in patients with conservatively managed spontaneous coronary artery dissection from the multicentre DISCO registry

Cited by 15 publications

References 0 publications

Balloon assisted hematoma fenestration in spontaneous coronary artery dissection. Case presentation and literature review

Balloon assisted hematoma fenestration in spontaneous coronary artery dissection. Case presentation and literature review

Myocardial infarction with nonobstructive coronary arteries: from pathophysiology to therapeutic strategies

Diseksi Spontan Arteri Koroner: Diagnosis dan Manajemen

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