Corrigendum to ‘Ginsenoside Rg1 promoted the wound healing in diabetic foot ulcers via miR-489e3p/Sirt1 axis’[Journal of Pharmacological Sciences 147 (2021) 271–283]

Huang, L J; Cai, Hongbin; Zhang, Mingsheng; Liao, Ruo-Yi; Huang, Xing; Hu, Feng-Dan

doi:10.1016/j.jphs.2022.03.006

Cited by 1 publication

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“…These findings imply that miR-133b negatively regulates angiogenesis during the proliferative phase of wound healing. Huang et al [ 47 ] found that miR-489-3p downregulation increases sirtuin (SIRT) 1 expression, promotes the PI3K/AKT/eNOS signaling pathway, improves cellular antioxidant capacity, and alleviates DFU. MiR-199a-5p has an important role in the development of diabetes and its complications[ 48 , 49 ].…”

Section: Mirnasmentioning

confidence: 99%

Non-coding RNAs: Role in diabetic foot and wound healing

Tang¹,

Uwimana²,

Zhu³

et al. 2022

World J Diabetes

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Diabetic foot ulcer (DFU) and poor wound healing are chronic complications in patients with diabetes. The increasing incidence of DFU has resulted in huge pressure worldwide. Diagnosing and treating this condition are therefore of great importance to control morbidity and improve prognosis. Finding new markers with potential diagnostic and therapeutic utility in DFU has gathered increasing interest. Wound healing is a process divided into three stages: Inflammation, proliferation, and regeneration. Non-coding RNAs (ncRNAs), which are small protected molecules transcribed from the genome without protein translation function, have emerged as important regulators of diabetes complications. The deregulation of ncRNAs may be linked to accelerated DFU development and delayed wound healing. Moreover, ncRNAs can be used for therapeutic purposes in diabetic wound healing. Herein, we summarize the role of microRNAs, long ncRNAs, and circular RNAs in diverse stages of DFU wound healing and their potential use as novel therapeutic targets.

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Section: Mirnasmentioning

confidence: 99%