Corrosive Ingestion

Abstract

Corrosive ingestion remains a common problem in developing countries, such as India due to the lack of strict laws that regulate the sale of caustics. While appropriate treatment of the acute phase can mitigate tissue damage improper management of the acute corrosive injury is widely prevalent due to the limited experience of the individual physicians in managing this condition. The aim of this review is to summarize the epidemiology and pathophysiology of corrosive ingestion, principles in the management of acute phase injury, long-term effects of caustic ingestion, and prevention of corrosive ingestion.

A case of severe upper alimentary tract necrosis by corrosive acid poisoning

“…Suicidal ingestion of corrosive substances will have predominant oral, pharyngeal, and upper oesophageal lesions, whereas accidental ingestion has predominant gastric injuries, although esophageal injuries may be associated. 3 Acids are known to cause coagulative necrosis of tissues.…”

Isolated jejunal necrosis following corrosive acid poisoning

“…Corrosive injuries of the upper gastrointestinal tract are frequently seen in developing countries like India. 2 It can be caused by acid or alkali ingestion. Suicidal attempts are common in adults.…”

“…1 Extensive injuries of the upper gastrointestinal tract can occur in patients who have consumed lethal amounts. 1,2 Isolated jejunal involvement is infrequent to occur. Patients usually present with abdominal pain, breathlessness, tachycardia, and vomiting.…”

Acid corrosive injury of stomach: management and long-term outcome

“…It usually starts within 2-3 weeks and progress rapidly. 5 Injury to stomach as a consequences of acid ingestion usually occur along lesser curvature and prepyloric region. Prepyloric and antrum is the commonest site owing to reflex pylorospasm and pooling of secretions in this region.…”