Course and Outcome of Fetuses Suspected of Having Coarctation of the Aorta during Gestation

Axt-Fliedner, R; Hartge, D.R.; Krapp, M.; Berg, Christoph; Geipel, A.; Koester, Stefan W.; Noack, F.; Germer, U.; Gembruch, Ulrich

doi:10.1055/s-2008-1027556

Cited by 27 publications

(15 citation statements)

References 9 publications

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“…The association of left–right ventricular disproportion with both cardiac and extracardiac pathology has been reported before by Hornung et al . and Axt‐Fliedner et al ,. who found a considerable incidence of other CHDs, such as VSD, PLSVC, pulmonary or aortic valve stenosis and chromosomal diseases in these patients.…”

Section: Discussionsupporting

confidence: 59%

Postnatal outcome of fetal isolated ventricular size disproportion in the absence of aortic coarctation

Nisselrooij

Rozendaal

Linskens

et al. 2018

Ultrasound in Obstet & Gyne

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The postnatal course of cases with fetal ventricular size disproportion is complicated by prenatally undetected congenital defects (46%) and pulmonary or transition problems (35%) in a significant number of cases that do not develop CoA. Proper monitoring of these cases is therefore warranted and it is advisable to incorporate the risks for additional morbidity and neonatal complications in prenatal counseling. Copyright © 2017 ISUOG. Published by John Wiley & Sons Ltd.

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Section: Discussionsupporting

confidence: 59%

Postnatal outcome of fetal isolated ventricular size disproportion in the absence of aortic coarctation

Nisselrooij

Rozendaal

Linskens

et al. 2018

Ultrasound in Obstet & Gyne

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“…Of the 28 neonates, 15 were male and 13 were female. The median gestational age at referral was 27 weeks (19)(20)(21)(22)(23)(24)(25)(26)(27)(28)(29)(30)(31)(32)(33). Postnatal echocardiography confirmed IAA in all 8 neonates with prenatal diagnosis.…”

Section: Resultsmentioning

confidence: 99%

“…The diagnosis of VSD is facilitated using color Doppler echocardiography. In contrast, if coarctation of the aorta is present, there is often a marked ventricular asymmetry, with the left side being smaller than the right side, unless there is a large VSD [22] . However, a significant discrepancy in great vessel size can be diagnosed in case of IAA on 2-DE and color Doppler in a transverse section of the upper mediastinum, the so-called 3-vessel view, usually used to visualize the aortic arch in transverse section.…”

Section: Discussionmentioning

confidence: 98%

Fetal and Neonatal Diagnosis of Interrupted Aortic Arch: Associations and Outcomes

et al. 2011

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Objective: Interrupted aortic arch (IAA) is a rare but serious anomaly. Prenatal diagnosis is challenging and published data are limited. The aim of the study was to review the data of fetuses and neonates diagnosed with IAA during a 16-year period at Children’s Hospital Giessen. Methods: Retrospective ascertainment of 8 fetuses and 20 neonates with a confirmed diagnosis of IAA from 1994 to 2010 by reviewing the hospital database of the cardiovascular program of the prenatal and pediatric cardiology clinics at the University Hospital Giessen. Results: Eighteen cases with IAA type B and 10 cases with IAA type A were found. After 2005, prenatal diagnosis was achieved in 8 cases and postnatal imaging confirmed IAA in all 8 neonates. Twenty-nine percent of individuals had a chromosomal anomaly, with microdeletion 22q11.2 being the most common abnormality (n = 6, 21%). In 46% (13/28) other complex cardiac anomalies were present. Mortality after surgery was 18%. Long-term morbidity and mortality was due to neurological impairment in the presence of microdeletion 22q11.2 and the need of surgical or catheter re-intervention. Conclusion: Despite the difficulties and challenges in diagnosis, the prenatal detection rate of IAA is increasing. Associated complex cardiac and chromosomal abnormalities influence the outcome of patients with IAA and are important issues of parental counseling.

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“…In those examples of HLH, the size of left heart dimensions is well below the normal values in mid-gestation [6, 14 -16]. In a smaller proportion of fetuses, the mitral and aortic valves are patent with either aortic arch hypoplasia, coarctation of the aorta or aortic valve stenosis [17]. It is known that those lesions might progress to HLH by birth [18,19].…”

Section: Zusammenfassung !mentioning

confidence: 99%

Prenatal Assessment of Ventriculocoronary Connections and Ventricular Endocardial Fibroelastosis in Hypoplastic Left Heart

et al. 2014

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The presence of VCAC in HLH can be diagnosed by fetal echocardiography predominantly occurring in cases with obstructed outflow and to some extent patent mitral valve. EFE is a frequent coexisting finding. Hospital survival was comparable among different anatomical subgroups and in cases with VCAC. The presence of VCAC in HLH did not limit the results of surgical palliation within the observation period of 30 days.

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Course and Outcome of Fetuses Suspected of Having Coarctation of the Aorta during Gestation

Cited by 27 publications

References 9 publications

Postnatal outcome of fetal isolated ventricular size disproportion in the absence of aortic coarctation

Postnatal outcome of fetal isolated ventricular size disproportion in the absence of aortic coarctation

Fetal and Neonatal Diagnosis of Interrupted Aortic Arch: Associations and Outcomes

Prenatal Assessment of Ventriculocoronary Connections and Ventricular Endocardial Fibroelastosis in Hypoplastic Left Heart

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