Coxsackie virus heart disease and cardiomyopathy

Levi, Guido; Proto, Claudia; Quadri, A; Ratti, Silvia G.

doi:10.1016/s0002-8703(77)80402-x

Cited by 35 publications

(11 citation statements)

References 14 publications

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“…It is conceivable that many alcoholics with DCM may have had a mild or forgotten viral infection and their chronic DCM may represent a modified form of burned out viral myocarditis [37]. About one third of patients with a past history of proven Coxsackie viral myocarditis show a depression of myocardial function 2–5 years after the acute episode [38]. In India about 15% of patients with proven viral myocarditis had persistent heart failure when seen 3 months later [39].…”

Section: Genuine Alcoholic Cardiomyopathies: Symptomatic Typesmentioning

confidence: 99%

The Alcoholic Cardiomyopathies – Genuine and Pseudo

Constant

1999

Cardiology

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Two types of genuine alcoholic cardiomyopathies can produce congestive heart failure, an acute fulminate hyperkinetic type with high cardiac output and a hypokinetic type with a low cardiac output, both of which respond to thiamine. When the chronic alcoholic develops a dilated cardiomyopathy with congestive heart failure without a history of significant malnutrition, then they do not respond to thiamine. This may be explained by a study in which mice given a myotropic virus plus alcohol developed more frequent and severe viral cardiomyopathies than those not fed alcohol. This suggests that a chronic dilated cardiomyopathy in an alcoholic may not be the direct result of the alcohol but secondary to a viral myocarditis. This suggests that the preferred terminology for a dilated cardiomyopathy in an alcoholic who does not respond to thiamine and good nutrition should simply be a dilated cardiomyopathy in an alcoholic. We should assume that the cardiomyopathy is possibly of viral etiology.

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Section: Genuine Alcoholic Cardiomyopathies: Symptomatic Typesmentioning

confidence: 99%

The Alcoholic Cardiomyopathies – Genuine and Pseudo

Constant

1999

Cardiology

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“…Although in several cases of acute viral myo-carditis progression towards cardiac enlargement and/or dysfunction has been noted (Sainani, Krompstie and Slodki, 1968;Smith, 1970;Levi et al, 1977), the authors believe that this is the first reported case of acute congestive cardiomyopathy complicating acute viral myocarditis. Furthermore, they document the first described case of varicella cardiomyopathy.…”

Section: Discussionmentioning

confidence: 76%

Varicella myocarditis producing congestive cardiomyopathy

Thandroyen

Asmal

Armstrong

1981

Postgraduate Medical Journal

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“…A significantly increased number of large mononuclear cells (one of the NK cell subpopulations) has been observed in the convalescent stages of myocarditis (Kanda et al 1990). Levi et al (1977) reported that patients with coxsackievirus myocarditis have a good prognosis. Another possibility is that damaged myocardium with intercellular fibrosis may have a reduced interstitial space and consequently a reduced capacity to manufacture lymphocytes.…”

Section: Discussionmentioning

confidence: 99%