Crisis disautonómicas tras traumatismo craneoencefálico grave

Fernández-Ortega, Juan Francisco; Prieto-Palomino, Miguel Angel; Muñoz-López, Alfonso; Hernández-Sierra, Bárbara; Seller-Pérez, G.; Quesada-García, Guillermo

doi:10.1016/s0210-5691(04)70084-4

Cited by 2 publications

(1 citation statement)

References 12 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…14 The most common symptoms of PSH are hyperthermia 23 ; excessive diaphoresis 12 ; posture in extension, 3 decerebrate, decortication, rigidity and spasticity 14 ; dystonia; tachycardia 7 ; excessive salivation; tachypnea; and arterial hypertension. 24 These signs and symptoms vary from episode to episode, as well as from individual to individual. 6 The interruption of diaphoresis is used as a mark between the second and third phases, frequently occurring on the 74 th day after the brain injury.…”

Section: Clinical Coursementioning

confidence: 99%

Paroxysmal Sympathetic Hyperactivity in Patients Victims of Traumatic Brain Injury: Literature Review

Kalkmann

Crestani

Santos

et al. 2021

Arquivos Brasileiros de Neurocirurgia: Brazilian Neurosurgery

View full text Add to dashboard Cite

The present literature review aims to present the physiology of paroxysmal sympathetic hyperactivity (PSH) as well as its clinical course, conceptualizing them, and establishing its diagnosis and treatment. Paroxysmal sympathetic hyperactivity is a rare syndrome, which often presents after an acute traumatic brain injury. Characterized by a hyperactivity of the sympathetic nervous system, when diagnosed in its pure form, its symptomatologic presentation is through tachycardia, tachypnea, hyperthermia, hypertension, dystonia, and sialorrhea. The treatment of PSH is basically pharmacological, using central nervous system suppressors; however, the nonmedication approach is closely associated with a reduction in external stimuli, such as visual and auditory stimuli. Mismanagement can lead to the development of serious cardiovascular and diencephalic complications, and the need for neurosurgeons and neurointensivists to know about PSH is evident in order to provide a fast and accurate treatment of this syndrome.

show abstract