Crystalline silica and lung cancer: A review of recent experimental evidence

Holland, L.M.

doi:10.1016/s0273-2300(05)80060-1

Cited by 24 publications

(12 citation statements)

References 11 publications

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“…The diversity of opinion and discrepancy of results show that this problem is very complex and, therefore, its definite solution is very difficult [Barisione et al, 1991;Heppleston, 1985;Holland, 1990;McDonald, 19891. The opinion of these authors as well as our own experience seem …”

Section: Discussionmentioning

confidence: 99%

Mortality among different occupational groups of workers with pneumoconiosis: Results from a register-based cohort study

Starzyński¹,

K²,

Kujawska³

et al. 1996

Am. J. Ind. Med.

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Section: Discussionmentioning

confidence: 99%

Mortality among different occupational groups of workers with pneumoconiosis: Results from a register-based cohort study

Starzyński¹,

K²,

Kujawska³

et al. 1996

Am. J. Ind. Med.

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“…Breast cancer is a malignant tumor that occurs in glandular epithelium of breasts and is regarded as the first major harm to women’s health 43 . In the predictive dataset, 78 miRNAs are related to breast cancer.…”

Section: Resultsmentioning

confidence: 99%

Improved low-rank matrix recovery method for predicting miRNA-disease association

Peng

Liao

Huang

et al. 2017

Sci Rep

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MicroRNAs (miRNAs) performs crucial roles in various human diseases, but miRNA-related pathogenic mechanisms remain incompletely understood. Revealing the potential relationship between miRNAs and diseases is a critical problem in biomedical research. Considering limitation of existing computational approaches, we develop improved low-rank matrix recovery (ILRMR) for miRNA-disease association prediction. ILRMR is a global method that can simultaneously prioritize potential association for all diseases and does not require negative samples. ILRMR can also identify promising miRNAs for investigating diseases without any known related miRNA. By integrating miRNA-miRNA similarity information, disease-disease similarity information, and miRNA family information to matrix recovery, ILRMR performs better than other methods in cross validation and case studies.

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“…1993;Peters-Golden et al, 1985;Roumm and Medsger, 1985 Idiopathic pulmonary Stereotyped inflammatory Hubbard et al, 2000;Park et al, 1999; fibrosis (cryptogenic response of alveolar wall to injury. Harris et al, 1998;Lee et al, 1996;Nagai fibrosing alveolitis) et al, 1992;Enomoto et al, 1985;Turner-Warwick et al, 1980 Silicosis Crystalline silica (e.g., quartz, sand) Holland, 1990;Tashiro et al, 1990;Amandus and Costello, 1991;Dong et al, 1995;Wang et al, 1996;Weill and McDonald, 1996;Brown et al, 1997 Subclinical infection Human Pneumocystis de la Horra et al, 2004 Table 3 lists studies reporting a positive association between benign lung diseases and lung cancer. Chronic bronchitis is a benign disease of the airways defined clinically by the chronic presence of cough and sputum production (Nadel, 2002).…”

Section: Chronic Inflammatory Lung Diseases Associated With Lung Cancermentioning

confidence: 99%

Perspectives on Pulmonary Inflammation and Lung Cancer Risk in Cigarette Smokers

Smith

Perfetti²,

King

2006

Inhalation Toxicology

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Cigarette mainstream smoke (MSS) inhaled by smokers is a complex aerosol composed of minute liquid droplets suspended within a mixture of combustion gases (CO, CO2, NOx, etc.) and semivolatile compounds. The minute liquid droplets represent the particulate or "tar" phase, while the combustion gases and semivolatiles comprise the vapor phase. For historical and technical reasons, the vast majority of studies on the carcinogenicity of MSS have focused on the particulate phase. The particulate phase is mutagenic and cytotoxic in vitro, proinflammatory, and promotes tumor formation in animal models. In addition to cytotoxic compounds found in the particulate phase, the vapor phase of MSS contains a number of cytotoxic constituents including reactive aldehydes and carbonyls capable of damaging cells and inducing pulmonary inflammation. A large body of evidence suggests that smoking-induced pulmonary inflammation may play an important role in increasing lung cancer risk in smokers. Use of aspirin and nonsteroidal anti-inflammatory drugs is associated with reduced cancer development in animal models and lower lung cancer rates in smokers. A number of benign nonpulmonary and pulmonary diseases characterized by chronic inflammation increase the risk of cancer at the affected site in the absence of chemical exposure. Animal models displaying tumorigenic responses following exposure to either whole smoke or smoke fractions show elevated rates of cellular proliferation. A relationship between pulmonary inflammation and lung cancer is mechanistically plausible because inflammatory cells secrete activated oxygen species, inflammatory mediators, and proteolytic enzymes that can both damage DNA and lead to increases in reparative cell proliferation rates.

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Crystalline silica and lung cancer: A review of recent experimental evidence

Cited by 24 publications

References 11 publications

Mortality among different occupational groups of workers with pneumoconiosis: Results from a register-based cohort study

Mortality among different occupational groups of workers with pneumoconiosis: Results from a register-based cohort study

Improved low-rank matrix recovery method for predicting miRNA-disease association

Perspectives on Pulmonary Inflammation and Lung Cancer Risk in Cigarette Smokers

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