Cutaneous blood flow and adrenoceptor response increase in segmental-type vitiligo lesions

Wu, Chieh-Shan; Yu, Hsin‐Su; Chang, Huoy-Rou; Yu, Chao; Yu, Chia‐Li; Wu, Bin–Nan

doi:10.1016/s0923-1811(99)00090-0

Cited by 70 publications

(69 citation statements)

References 33 publications

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“…Ach has an inhibitory effect on dopa oxidase activity in both dendritic and nondendritic marginal melanocytes in vitiligo. It is therefore reasoned that a fall in AchE activity in the melanocytes leads to greater inhibition by Ach, aggravating the process of depigmentation in vitiligo [4].In summary, increased NE and Ach levels, and increased activation of · and ß receptors [5], seem to play important roles in the pathogenesis of vitiligo. …”

mentioning

confidence: 99%

Prescribing Cyclic Antidepressants for Vitiligo Patients: Which Agents Are Superior, Which Are Not?

Namazi¹

2003

Psychother Psychosom

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mentioning

confidence: 99%

Prescribing Cyclic Antidepressants for Vitiligo Patients: Which Agents Are Superior, Which Are Not?

Namazi¹

2003

Psychother Psychosom

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“…20 However, NSV results from progressive autoimmune loss of melanocytes in the involved area. To determine that genetic factors, especially variation in the TXNDC5 gene, contribute to melanocyte destruction, we focused on NSV.…”

Section: Discussionmentioning

confidence: 99%

Association of TXNDC5 gene polymorphisms and susceptibility to nonsegmental vitiligo in the Korean population

Jeong¹,

Shin²,

Uhm³

et al. 2009

British Journal of Dermatology

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“…In SV, the neural/neurogenic theory supported by clinical, ultrastructural and biochemical arguments was reported [8,9]. However data are discordant and the role of nervous system is probable but poorly established.…”

Section: Sv and Nsv As Separate Entitiesmentioning

confidence: 99%

New Insights in Mixed Vitiligo: Initial Non Segmental Vitiligo can precede the Onset of Segmental Vitiligo (Cases Report and Review of Theories)

Benzekri¹,

Gauthier²

2014

Pigmentary Disorders

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Recently, mixed vitiligo (MV) was defined by Mulekar as the association of segmental vitiligo (SV) and nonsegmental vitiligo (NSV) in the same patient. Until now, SV and generalized NSV were considered to be separate entities with a different distribution (unilateral or bilateral). Generalized vitiligo seemed to be more frequently associated with other autoimmune diseases than SV. From the pathophysiological standpoint, it was hypothesized that NSV was an autoimmune disease, while SV may result of a sympathetic dysfunction. The first pediatric case of SV associated with NSV was reported in 2003 and 36 other observations were subsequently reported. In all these cases, segmental involvement associated usually in a second step with the onset of bilateral vitiligo patches. In this article, we describe, for the first time, two atypical cases of mixed vitiligo in which NSV involvement preceded the onset of SV. In the light of these remarkable cases, different possible aetiopathogenetic mechanisms of MV are discussed which may provide new insights into the pathogenesis of vitiligo.

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Cutaneous blood flow and adrenoceptor response increase in segmental-type vitiligo lesions

Cited by 70 publications

References 33 publications

Prescribing Cyclic Antidepressants for Vitiligo Patients: Which Agents Are Superior, Which Are Not?

Prescribing Cyclic Antidepressants for Vitiligo Patients: Which Agents Are Superior, Which Are Not?

Association of TXNDC5 gene polymorphisms and susceptibility to nonsegmental vitiligo in the Korean population

New Insights in Mixed Vitiligo: Initial Non Segmental Vitiligo can precede the Onset of Segmental Vitiligo (Cases Report and Review of Theories)

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