Das Einschlaferleben und seine neurophysiologischen Korrelate

Kuhlo, W; Lehmann, Dietrich

doi:10.1007/bf00344881

Cited by 55 publications

(11 citation statements)

References 14 publications

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“…Aserinsky and Kleitman (2) described slow eye movements (SEMs) during drowsiness preceding sleep onset and during light sleep. Kuhlo and Lehmann (28) found that SEMs became larger and more regularly sinusoidal when simultaneous slowing of the EEG was noted during sleep onset. Slow (0.25 Hz), pendular, horizontal eye movements were seen as the first sign of drowsiness in 50.5% of the 200 US Air Force flight personnel in a field study by Maulsby et al (32).…”

mentioning

confidence: 99%

EEG and ocular correlates of circadian melatonin phase and human performance decrements during sleep loss

Cajochen

Khalsa

Wyatt

et al. 1999

American Journal of Physiology-Regulatory, Integrative and Comp

240

252

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The aim of this study was to quantify the associations between slow eye movements (SEMs), eye blink rate, waking electroencephalogram (EEG) power density, neurobehavioral performance, and the circadian rhythm of plasma melatonin in a cohort of 10 healthy men during up to 32 h of sustained wakefulness. The time course of neurobehavioral performance was characterized by fairly stable levels throughout the first 16 h of wakefulness followed by deterioration during the phase of melatonin secretion. This deterioration was closely associated with an increase in SEMs. Frontal low-frequency EEG activity (1–7 Hz) exhibited a prominent increase with time awake and little circadian modulation. EEG alpha activity exhibited circadian modulation. The dynamics of SEMs and EEG activity were phase locked to changes in neurobehavioral performance and lagged the plasma melatonin rhythm. The data indicate that frontal areas of the brain are more susceptible to sleep loss than occipital areas. Frontal EEG activity and ocular parameters may be used to monitor and predict changes in neurobehavioral performance associated with sleep loss and circadian misalignment.

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mentioning

confidence: 99%

EEG and ocular correlates of circadian melatonin phase and human performance decrements during sleep loss

Cajochen

Khalsa

Wyatt

et al. 1999

American Journal of Physiology-Regulatory, Integrative and Comp

240

252

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“…(1971), which was suggested by Rotenberg (1982) and another by Schwartz (1968), which was mentioned by Gottesmann, were not referred to in my target article but contain evidence fairly directly supporting the covert REM sleep model. He also cites a study by Kuhlo and Lehmann (1964) in which eye movements sim-ilar to his medium fast eye movements were studied in conjunction with hypnagogic imagery. Toth (1971) devised miniature electrodes which, when glued to the eyelids overlying the cornea, more than doubled the sensitivity of standard EEG recordings.…”

Section: Nr1 the Definition Of Dreaming Is Inadequatementioning

confidence: 99%

REM sleep deprivation: The wrong paradigm leading to wrong conclusions

Born¹,

Gais²

2000

Behav Brain Sci

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There are obvious flaws in REM sleep suppression paradigms that do not allow any conclusion to be drawn either pro or contra the REM sleep-memory hypothesis. However, less intrusive investigations of REM sleep suggest that this sleep stage or its adjunct neuroendocrine characteristics exert a facilitating influence on certain aspects of ongoing memory formation during sleep.[Nielsen; Vertes & Eastman]

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“…und sind als isoliertes Symptom nicht krankhaft (58,108). Häufiger sind sie ein Teil eines narkoleptischen Syndromes.…”

Section: G Hajak B Bandel(jwunclassified

Koinzidenz von Angst- und Schlafstörungen

Hajak¹,

Bandelow²

1996

Fortschr Neurol Psychiatr

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Anxiety disorders and sleep disorders show various interrelations. This paper stresses the clinical importance of sleep problems in relation to anxiety disorders and of anxiety symptoms in patients with sleep disorders, in context with the relevant literature. Differential diagnostic aspects of overlapping of anxiety, sleep disturbances and organic diseases with anxiety-like symptoms are presented. Sleep-related panic attacks and anxiety in insomnia provide examples of the development of pathophysiological concepts coming from the coupling of sleep and anxiety. Case reports illustrate the relevance for clinical practice.

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Das Einschlaferleben und seine neurophysiologischen Korrelate

Cited by 55 publications

References 14 publications

EEG and ocular correlates of circadian melatonin phase and human performance decrements during sleep loss

EEG and ocular correlates of circadian melatonin phase and human performance decrements during sleep loss

REM sleep deprivation: The wrong paradigm leading to wrong conclusions

Koinzidenz von Angst- und Schlafstörungen

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