2021
DOI: 10.3390/life11020108
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Dectin-1-Mediated DC-SIGN Recruitment to Candida albicans Contact Sites

Abstract: At host–pathogen contact sites with Candida albicans, Dectin-1 activates pro-inflammatory signaling, while DC-SIGN promotes adhesion to the fungal surface. We observed that Dectin-1 and DC-SIGN collaborate to enhance capture/retention of C. albicans under fluid shear culture conditions. Therefore, we devised a cellular model system wherein we could investigate the interaction between these two receptors during the earliest stages of host–pathogen interaction. In cells expressing both receptors, DC-SIGN was qui… Show more

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Cited by 3 publications
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