Deep Vein Thrombosis of the Lower Extremity in a Football Player: A Case Report

Thompson, Terry L.; Robinson, Audrey K.; Gilbert, Corey

doi:10.1097/00042752-200607000-00019

Cited by 5 publications

(2 citation statements)

References 10 publications

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“…DVT in such circumstances may not be a rare occurrence as a direct consequence of sport-induced trauma [3]. However, very few cases of lower extremity DVT involving direct sport-associated trauma are reported in the medical literature [5][6][7][8][9]. From these, there is one case report that specifically related DVT development to soccer-related trauma [5], and a case of a 42 year-old male semi-professional soccer player who sustained a right lower extremity popliteal contusion during a soccer game resulting in a traumatic DVT [6].…”

Section: Discussionmentioning

confidence: 99%

Deep venous thromboembolism after a trauma in a football player double heterozygous for factor V Leiden and prothrombin G20210A mutation: The role of genetic testing in sport

Botto

Pasanisi

Chubuchny

et al. 2012

Journal of Cardiology Cases

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Traumatic vascular injuries to the lower limb are frequent in athletes, particularly in sports characterized by high-speed collisions. However, the diagnosis is not always straightforward, for the lack of clearly visible abnormalities without provocative testing or appropriate imaging. The failure of an early diagnosis can lead to devastating consequences. In these subjects, it may be useful to investigate the personal susceptibility to thrombotic events such as the presence of a hereditary hypercoagulable state. We experienced a case of a soccer player with progressive swelling and severe pain of the calf after a trauma during a football match 3 days previously, who came to our hospital for suspected deep vein thrombosis, confirmed by echo-Doppler ultrasound. A thrombophilia screening detected a double heterozygosity for factor V Leiden and prothrombin G20210A mutation in the presence of a strong family history for thromboembolism. Immediate treatment with elastic stocking compression and enoxaparin was started. The patient was discharged on warfarin therapy maintained for six months, with the warning to avoid trauma activities during anticoagulation. Thrombotic genetic testing in athletes who experience episodes of deep vein thrombosis might offer important opportunities for patient management, such as prolonged anticoagulant therapy or avoidance of risk factors such as trauma-related sports.

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Section: Discussionmentioning

confidence: 99%

Deep venous thromboembolism after a trauma in a football player double heterozygous for factor V Leiden and prothrombin G20210A mutation: The role of genetic testing in sport

Botto

Pasanisi

Chubuchny

et al. 2012

Journal of Cardiology Cases

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“…Abnormal congenital congealing fibrinogenolysis; drug use, including use of contraceptives and cocaine; immobilization, such as prolonged sitting; and malignant tumors are the most common causes of DVT [1][2][3][4]. Furthermore, a few cases of sport-related lower extremity DVT, involving direct external trauma, have been reported [5][6][7]…”

Section: Introductionmentioning

confidence: 99%

Acute pulmonary thromboembolism from deep vein thrombosis induced by trauma to the popliteal vein with a tennis racket

Ise

Igarashi

Masuda

et al. 2014

Journal of Cardiology Cases

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a b s t r a c tAcute pulmonary thromboembolism (PTE) is mainly caused by deep vein thrombosis (DVT) and sometimes leads to a fatal outcome. Few cases of sport-related lower extremity DVT, involving direct external trauma, have been reported. A 58-year-old man, without any risk factors for thromboembolism suffered acute PTE from DVT after playing tennis. A detailed history revealed that he had hit his popliteal vein with each swing of his tennis racket and the site of the trauma, at popliteal fossa, was exactly the same as the site of the DVT formation. Therefore, the cause of DVT was suspected to be the repeated trauma to the popliteal vein. The repeated external trauma to the popliteal vein may have caused vascular endothelial damage, leading to DVT.

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