1997
DOI: 10.1097/00003246-199706000-00013
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Delayed hyperemia causing intracranial hypertension after cardiopulmonary resuscitation

Abstract: Delayed hyperemia occurs in humans after resuscitation from cardiac arrest. Our data suggest that this delayed hyperemia can lead to intracranial hypertension and occasionally acute brain swelling, contributing to a poor outcome. A high mean flow velocity of the middle cerebral artery with a low ratio of mean flow velocity of the middle cerebral artery to mean flow velocity of the internal carotid artery may be predictive of critical hyperemia. As an indirect method of measuring cerebral blood flow transcrania… Show more

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Cited by 77 publications
(40 citation statements)
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“…In indirect support of this assumption is the fact that the lower limit could be identified at a higher arterial pressure in 5 of the 13 patients. Conversely, there is no evidence of generally increased intracranial pressure shortly after resus- citation, 1,6,21,22 , whereas there is an increase in the number of patients with increased intracranial pressure in the days after resuscitation. 23 To conclude that the lower limit of autoregulation was identical in control subjects and patients, intracranial pressure should have been increased on average to 40 mm Hg, which seems unlikely.…”
Section: Discussionmentioning
confidence: 94%
“…In indirect support of this assumption is the fact that the lower limit could be identified at a higher arterial pressure in 5 of the 13 patients. Conversely, there is no evidence of generally increased intracranial pressure shortly after resus- citation, 1,6,21,22 , whereas there is an increase in the number of patients with increased intracranial pressure in the days after resuscitation. 23 To conclude that the lower limit of autoregulation was identical in control subjects and patients, intracranial pressure should have been increased on average to 40 mm Hg, which seems unlikely.…”
Section: Discussionmentioning
confidence: 94%
“…Intracranial hypertension has been shown to occur late in post-arrest patients, potentially due to hyperemic perfusion. 15 The cause for cardiac arrest may also contribute to the degree of cerebral edema and HIBI. Arrests caused by respiratory failure cause cerebral hypoxia before the cessation of cardiac function, potentially worsening the degree of brain injury compared to ischemic arrests.…”
Section: Discussionmentioning
confidence: 99%
“…976 The timing of transcranial Doppler in this study ranged from 4 to 120 hours after ROSC. Transcranial Doppler parameters associated with poor outcome included delayed hyperemia.…”
Section: S378mentioning
confidence: 99%