Dens in dente

Shafer, William G.; Hine, Maynard K.

doi:10.1016/0030-4220(52)90130-8

Cited by 180 publications

(298 citation statements)

References 5 publications

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“…Many explanations for the etiopathogenesis of taurodontism have been put forward from time to time. These include: a) an unusual developmental delay; b) delay in calcification of the pulp chamber floor; c) odontoblastic deficiency (d) alteration in hertwig's epithelial root sheath, with an apparent failure of the epithelial diaphragm to invaginate at the normal horizontal level and e) delayed or incomplete union of the horizontal flaps of the epithelial diaphragm [7].…”

Section: Discussionmentioning

confidence: 99%

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Non Syndromic Familial Bilateral Decidious Taurodontism – A First Case Report

Panigrahi

Srilatha

et al. 2014

JCDR

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A 4-year-old male child escorted by his mother, elder brother and sister had come to the clinic with the chief complaint of painful decayed teeth in right and left lower jaw since two weeks. Pain was intermittent, mild and non-radiating in nature.His medical history was non-contributory.General and intra-oral soft tissue examination revealed normal appearance. Intraoral hard tissue examination revealed the presence of following teeth. On clinical examination there was deep carious lesion in relation to 74 and 84 and grossly destroyed 64 and 85. Intraoral periapical radiograph of 74 and 84 revealed radiolucency involving pulp and enlarged pulp chamber with short roots suggestive of taurodontism [Table/ Fig-1,2]. The teeth were subjected for clinical tests and diagnosis of chronic irreversible pulpitis was made in relation to 64, 74, 84, and 85. The teeth 74 and 84 were treated by multivisit pulpectomy procedure. Obturation was done with vitapex followed by semi permanent restoration with stainless steel crowns [ Fig-7] of sister exhibited taurodontic characteristic. Suspecting this the mother was also subjected to radiographic procedure who to our great surprise exhibited taurodontic appearance of a previously endodontically treated 36 [Table /Fig-8]. Dentistry SectionNon Syndromic Familial Bilateral Decidious Taurodontism -A First Case Report ABSTRACTTaurodontism is anatomical and morphological alteration in the shape of the tooth. In taurodontism the crowns appear larger and the roots shorter compared to the normal anatomy of the respective tooth. The incidence of taurodontism in deciduous is very low and is also not very common in permanent dentition. The endodontic treatment protocol of taurodontic tooth is challenging. Taurodontic tooth requires a very cautious approach in identifying the root canal orientation and presence of any extra or lateral canals.

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Section: Discussionmentioning

confidence: 99%

“…Usually, it is an isolated anomaly, but can occur in several well-known syndromes such as Down's syndrome, Klienfelter's syndrome, tricho-dento-osseous syndrome, Mohr syndrome, Wolf-Hirschhorn syndrome and Lowe syndrome [7][8][9].…”

Section: Discussionmentioning

confidence: 99%

Non Syndromic Familial Bilateral Decidious Taurodontism – A First Case Report

Panigrahi

Srilatha

et al. 2014

JCDR

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“…3 Marfan syndrome is a spectrum of disorders caused by a heritable genetic defect of connective tissue that has an autosomal dominant mode of transmission. 4 MFS is a heritable disorder where abnormalities of fibrillin protein are encoded by the fibrillin-1 gene (FBN1), a large gene JAYPEE Cardiovascular manifestations include mitral valve prolapse, mitral valve regurgitation, aortic dilatation, aortic regurgitation, acute aortic aneurysm with or without regurgitation, tricuspid valve and pulmonary valve regurgitation. 6,8 Between 75 and 85% of Marfan patients have loose or 'floppy' mitral valves, which are the valves that separate the chambers of the heart.…”

Section: Discussionmentioning

confidence: 99%

“…Skull radiographs (anterioposterior views and lateral views) may demonstrate a high arched palate, increased skull height and enlarged frontal sinus. 4 The diagnosis of MFS with classical phenotype remains largely clinical. The diagnosis is made by taking a family history and thorough examination of the patient's eyes, heart and bone structure.…”

Section: Discussionmentioning

confidence: 99%

Marfan Syndrome: A Report of Two Cases and Review

K¹,

T²,

Koteeswaran³

et al. 2011

JIAOMR

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Marfan syndrome is an autosomal dominant disorder of the connective tissues, resulting in abnormalities of the musculoskeletal system, cardiovascular system and eyes. It has a prevalence of 1 in 100,000 population and occurs in all ethnic groups. It may be familial or due to new mutation (30%), in the fibrillin gene on arm of chromosome 15. It is estimated that one person in every 3000 to 5000 has Marfan's syndrome may have cardiovascular abnormalities and may be complicated by infective endocarditis. About 90% of Marfan patients will develop cardiac complications.

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“…adhering to a tooth and producing acids that decalcify and undermine the enamel. 38 As the decay develops further, passing through the dentin toward the pulp, sensitivity is noted by the patient. A deep carious lesion may require endodontic (root canal) therapy.…”

Section: )mentioning

confidence: 99%