Dental erosion in a partially edentulous patient with gastroesophageal reflux disease: a case report

Picoș, Andrei; Nicoara, Petra; Crăiţoiu, Monica Mihaela

doi:10.15386/cjmed-388

Cited by 8 publications

(14 citation statements)

References 4 publications

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“…Acidic and sweet drinks and foods either directly cause erosion or decay with acid reflux by passing to the mouth or proximal esophagus or indirectly by leading to reflux attack (20).…”

Section: Clinical Featuresmentioning

confidence: 99%

From Newborn to Teenage: Gastroesophageal Reflux

Urgancı¹,

Usta²

2016

JAREM

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Gastroesophageal reflux (GER) is characterized by the involuntary passage of gastric contents into the esophagus. GER disease is defined as a persistent or intermittent passage of gastric contents into the esophagus, which often results in overt clinical signs and symptoms and affects the quality of life. GER is a common disorder in childhood and has a good prognosis. GER in infancy begins within the first month of life, peaks at the 4 th month, and after the first year, it steadily decreases and finally resolves at the age of 2 years. GER physiologically occurs due to the relaxation of the lower esophageal sphincter, independent of swallowing. The mechanisms against GER include the following; 1. Lower esophageal sphincter dysfunction, 2. Clearance effect of esophagus, 3. Esophageal mucosal integrity, and 4. Gastric emptying, which prevent the development of GER disease. Although the physiopathology of GER is still not clearly established, proposed factors are genetic, environmental, anatomic, hormonal, and neurogenic. The most responsible factor is lower esophageal sphincter relaxations. The clinical signs of GER varies with age but commonly appear with gastrointestinal and respiratory symptoms. The diagnosis of GER disease is based on history, physical examination, esophageal pH monitoring, multichannel intraluminal impedance and esophageal manometry, motility examinations, endoscopy, biopsy, examinations with barium, and nuclear scintigraphy. Treatment of GER includes non-pharmacological and pharmacological therapies, such as prokinetic agents and acid suppressors. Surgical treatment indications are relatively limited. (JAREM 2016; 6: 67-73)

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“…Acidic and sweet drinks and foods either directly cause erosion or decay with acid reflux by passing to the mouth or proximal esophagus or indirectly by leading to reflux attack (20).…”

Section: Clinical Featuresmentioning

confidence: 99%

From Newborn to Teenage: Gastroesophageal Reflux

Urgancı¹,

Usta²

2016

JAREM

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“…Studies have shown that up to 60 % of patients with dental erosions had pathological levels of acid refl ux when assessed by ambulatory esophageal pH monitoring [ 3 ]. Dental erosions as a manifestation of GERD have not only been seen in adults but also in children.…”

Section: Epidemiologymentioning

confidence: 99%

“…The causes may consist of extrinsic factors like acidic foods, beverages, or medications. However, there can also be intrinsic factors, the most common being GERD with regurgitation of gastric acid into the oral cavity [ 3 ].…”

Section: Introductionmentioning

confidence: 99%

Prevention and Control of Dental Erosion: Gastroesophageal Reflux Disease Management

Patel

Brady

2015

Dental Erosion and Its Clinical Management

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Approximately 10-20 % of the general population suffers from gastroesophageal refl ux disease or GERD. GERD can manifest as esophageal and extraesophageal symptoms. GERD may damage the dental tissues, causing disorders such as dental erosion. According to studies, 24 % of patients with GERD have dental erosion, and 32 % of adults and 17 % of children with dental erosion have GERD. However, not all affected persons will have classic symptoms of GERD. Dentists may be the fi rst persons to diagnose GERD in these "silent refl uxers," particularly when observing unexplained tooth erosion. The cause of GERD is multifactorial, but the basic cause is incompetent antirefl ux barriers at the gastroesophageal junction. However, other causes have also been attributed to GERD which include decreased saliva production, diet, eating habits, medications, and obesity. Typical manifestations of GERD are heartburn, regurgitation, and dysphagia. Other symptoms have also been associated with GERD, and if any "alarm symptoms" are present, then further evaluation is required. The location of the erosive tooth wear in the dentition is specifi c to each etiologic factor. Refl uxed acid fi rst damages the palatal surface of the upper incisors then the other surfaces of the maxillary teeth. In chronic GERD, the labial or buccal surfaces are affected then the occlusal surfaces of maxillary and mandibular teeth. The diagnosis of GERD can be made clinically, physiologically, anatomically, or functionally depending on the testing modality. The mainstay management

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“…Dental erozyonlar ile GÖRH ilişkisi kurulmuştur, ağıza ya da proksimal özofagusa asidik reflü olması ile gelişir (14).…”

Section: Rġsk Faktörlerġ Ve Patofġzyolojġunclassified