Development of Left Ventricular Outflow Obstruction in Idiopathic Hypertrophic Subaortic Stenosis

Rg, Williams; Rc, Ellison; As, Nadas

doi:10.1056/nejm197304262881702

Cited by 18 publications

(3 citation statements)

References 3 publications

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“…Hypercontractility in ASH Hypertrophic cardiomyopathy has been reported to be hypercontractile at rest (Bonow et al, 1981;Braunwald et al, 1964;Williams et al, 1973). Safar et al (1979) reported that in patients with borderline hypertension, preponderance of ASH and increased IVST/PWT ratio were found, and ASH could not be directly related to the level of blood pressure.…”

Section: Discussionmentioning

confidence: 99%

Difference in the response to isoproterenol between asymmetric septal hypertrophy and symmetric hypertrophy in patients with hypertrophic cardiomyopathy

Iida

Sugishita

Matsuda

et al. 1986

Clinical Cardiology

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Summary:The response to isoproterenol was studied in 9 patients with hypertrophic cardiomyopathy (HCM) and asymmetric septal hypertrophy (ASH), 9 patients with HCM and symmetric hypertrophy (SH), and 9 normal controls (NC), using digitized M-mode echocardiography. There was no significant difference in fractional shortening (FS) between ASH and SH, nor between SH and NC before isoproterenol infusion. During isoproterenol infusion, however, FS was significantly greater in ASH (60f6%) than in SH (53f7%) and NC (49f5%) (p

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Section: Discussionmentioning

confidence: 99%

Difference in the response to isoproterenol between asymmetric septal hypertrophy and symmetric hypertrophy in patients with hypertrophic cardiomyopathy

Iida

Sugishita

Matsuda

et al. 1986

Clinical Cardiology

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“…This is of particular interest, for experimentally the placenta is relatively impermeable to thyroxine, tri-iodothyronine, and TSH (Fisher, 1975). Does a hyperkinetic or hypertrophic circulatory state precede obstructive hypertrophic changes as Perloff (1971) and Williams et al (1973) have suggested? Is this change initiated in fetal life during rapid development when the heart muscle may be vulnerable to any physiological or pathological stimuli having inotropic and chronotropic effects?…”

Section: Cardiomyopathy and Thyroid Diseasementioning

confidence: 99%

Thyroid heart disease.

Symons¹

1979

Heart

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“…1964Raghib et al 1964Tafur et al 1964Rubhe et ai. 1965Nasser et al 1967Harris et al 1968Shand et al 1971Tajik et al 1971Barr et al 1973Pernot et al 1973Williams et al 1973 1 1 20 ? Physical examination and hospital course.…”

Section: Case Reportsmentioning

confidence: 99%

Asymmetric septal hypertrophy in a large Amish kindred

Dillon

Hurwitz

1975

Clinical Genetics

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We report a large Amish kindred in which asymmetric septal hypertrophy (ASH) appeared to segregate as an autosomal dominant gene. This kindred was ascertained through a 3–month‐old infant and her father. The striking differences between father and daughter regarding clinical presentation and course exemplify the variable expressivity of this condition. The importance of genetic factors in diagnosis and counseling are discussed. Previous reports of ASH in infants and children are reviewed.

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Development of Left Ventricular Outflow Obstruction in Idiopathic Hypertrophic Subaortic Stenosis

Cited by 18 publications

References 3 publications

Difference in the response to isoproterenol between asymmetric septal hypertrophy and symmetric hypertrophy in patients with hypertrophic cardiomyopathy

Difference in the response to isoproterenol between asymmetric septal hypertrophy and symmetric hypertrophy in patients with hypertrophic cardiomyopathy

Thyroid heart disease.

Asymmetric septal hypertrophy in a large Amish kindred

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