Diagnostic accuracy of cardiac markers for myocardial damage after radiofrequency catheter ablation

Hirose, Hideo; Kato, Koichi; Сузукі, Осаму; Yoshida, Tetsuro; Oguri, Mitsutoshi; Yajima, Kazuhiro; Hibino, Takeshi; Yokoi, Kiyoshi

doi:10.1007/s10840-006-9034-4

Cited by 22 publications

(26 citation statements)

References 23 publications

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“…Similar elevations in troponin T have also been demonstrated, but correlation to specific parameters of the electrophysiological procedure is less consistent (321,322). In all of these studies, patients suffered no complications requiring further intervention.…”

Section: Appendix 1 Relevant Author Relationships With Industry and mentioning

confidence: 60%

ACCF 2012 Expert Consensus Document on Practical Clinical Considerations in the Interpretation of Troponin Elevations

Newby

Jesse²,

Babb³

et al. 2012

Journal of the American College of Cardiology

347

134

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Section: Appendix 1 Relevant Author Relationships With Industry and mentioning

confidence: 60%

ACCF 2012 Expert Consensus Document on Practical Clinical Considerations in the Interpretation of Troponin Elevations

Newby

Jesse²,

Babb³

et al. 2012

Journal of the American College of Cardiology

347

134

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“…In our study, the higher cTnI and CK-MB levels in cryoballoon group may be explained by longer energy delivery duration compared to the other groups. Previous AF ablation studies have found a correlation between levels of cTnT and RF delivery duration and cumulative RF energy [24,27,28], but no data exist regarding other energy sources. Apart from the energy delivery duration, other factors may contribute to higher degree of myocardial injury in cryoablation, as suggested by increased levels of biomarkers post ablation.…”

Section: Previous Studiesmentioning

confidence: 99%

Biomarkers of myocardial injury with different energy sources for atrial fibrillation catheter ablation

Casella

Russo

et al. 2014

Cardiol J

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“…However, RFCA causes some myocardial damage at the site where the catheter tip contacts tissue. This damage may include lipid membrane disruption and metabolic and structural protein inactivation and denaturation, as well as nuclear damage (99 ). In a study of 60 patients undergoing RFCA who had no underlying structural heart disease, all patients were found to have increased postprocedure cardiac cTn, with all measurements exceeding the diagnostic threshold for MI (100 ).…”

Section: Other Medications Wallace Et Al (143 )mentioning

confidence: 99%

Increases of Cardiac Troponin in Conditions other than Acute Coronary Syndrome and Heart Failure

2009

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Background: Although cardiac troponin (cTn) is a cornerstone marker in the assessment and management of patients with acute coronary syndrome (ACS) and heart failure (HF), cTn is not diagnostically specific for any single myocardial disease process. This narrative review discusses increases in cTn that result from acute and chronic diseases, iatrogenic causes, and myocardial injury other than ACS and HF.Content: Increased cTn concentrations have been reported in cardiac, vascular, and respiratory disease and in association with infectious processes. In cases involving acute aortic dissection, cerebrovascular accident, treatment in an intensive care unit, and upper gastrointestinal bleeding, increased cTn predicts a longer time to diagnosis and treatment, increased length of hospital stay, and increased mortality. cTn increases are diagnostically and prognostically useful in patients with cardiac inflammatory diseases and in patients with respiratory disease; in respiratory disease cTn can help identify patients who would benefit from aggressive management. In chronic renal failure patients the diagnostic sensitivity of cTn for ACS is decreased, but cTn is prognostic for the development of cardiovascular disease. cTn also provides useful information when increases are attributable to various iatrogenic causes and blunt chest trauma.Summary: Information on the diagnostic and prognostic uses of cTn in conditions other than ACS and heart failure is accumulating. Although increased cTn in settings other than ACS or heart failure is frequently considered a clinical confounder, the astute physician must be able to interpret cTn as a dynamic marker of myocardial damage, using clinical acumen to determine the source and significance of any reported cTn increase.

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Diagnostic accuracy of cardiac markers for myocardial damage after radiofrequency catheter ablation

Abstract: h-FABP is an insensitive and less specific marker of myocardial damage in RFCA much along the lines of CK-MB and when compared with the proven accuracy of cTnT.

Cited by 22 publications

References 23 publications

ACCF 2012 Expert Consensus Document on Practical Clinical Considerations in the Interpretation of Troponin Elevations

ACCF 2012 Expert Consensus Document on Practical Clinical Considerations in the Interpretation of Troponin Elevations

Biomarkers of myocardial injury with different energy sources for atrial fibrillation catheter ablation

Increases of Cardiac Troponin in Conditions other than Acute Coronary Syndrome and Heart Failure

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