Die Magenwandnekrose nach selektiv-proximaler Vagotomie

Abstract

A personal observation and the analysis of the pertinent literature form the basis of this comprehensive report of gastric wall necrosis (GWN) following selective proximal vagotomy (SPV). The incidence of this specific and ischemic sequel of SPV is 0.25%. GWN carries specific morphological criteria, shows certain predisposing risk factors of general and local type, characteristic symptoms and well defined types of clinical courses. Technical failures and delay of operative therapy contribute mainly to the present mortality of approximately 50%. The efficacy of prophylactic measures appears to be limited, whereas immediate and adequate reoperation yields a good prognosis.

Gastric necrosis and perforation as a complication of splenectomy. Case report and related references

“…One possible explanation for such an occurrence is the existence of anatomical variations in the distribution of arterial supply to the stomach, creating a predisposition towards the appearance of potentially ischemic areas (1,14) . Other factors contributing towards the onset of gastric necrosis after splenectomy include arteriosclerosis, diabetes, renal insufficiency, uremia, cardiac insufficiency and the use of corticosteroids and other immunosuppressors (7,18) . Surgical procedures on the stomach itself or lower esophagus may also provoke gastric necrosis.…”

Acute Gastric Dilatation: “A Gluttony or a Crises in a Pandemic” An Update with review of Current literature

“…Anterior perforations of the gastric wall are more common than posterior ones. The site of perforation is dependent on the underlying disease state like peptic ulcer location, atherosclerosis, uremia, and the use of corticosteroids and immunosuppressive drugs [27], [30] . The underlying anatomical variations of arterial arcade to the stomach also predispose to ischemic and necrosis [26], [29] .…”