Diffuse muscular hypertrophy of esophagus

Iyer, Swaminath K.; Chandrasekhara, Kota L.; Sutton, A. L.

doi:10.1016/0002-9343(86)90627-3

Cited by 17 publications

(8 citation statements)

References 5 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…As in three of our cases an association with pyloric stenosis has been There has been much confusion in the literature over the terminology of this condition, with some authors using the terms 'leiomyomatosis' and 'diffuse hypertrophy' synonymously, and others preferring to separate diffuse thickening without nodules ('hypertrophy') from diffuse thickening with a confluent nodular pattern ('oesophageal Ieiomyomatosis') 22. However, these two patterns may co-exist in the same ~a t i e n t~.~~ (as in our case 3 ) or in different members of the same familyy, suggesting that they are different aspects of the same condition. The tendency to form nodules is perhaps related to the chronicity of the hyperplasia, analogous to noted5.…”

Section: Discussionmentioning

confidence: 80%

Familial oesophageal leiomyomatosis and nephropathy

et al. 1992

View full text Add to dashboard Cite

Four female members of the same family suffering from a rare combination of oesophageal leiomyomatosis and an Alport-like nephropathy are described. The disease is characterized by marked thickening of the oesophageal wall, usually also involving the proximal stomach, with or without discernible leiomyomatous nodule formation. All cases were treated surgically by oesophagectomy with symptomatic relief, and there was no evidence of recurrence on follow-up (2-37 years). The syndrome appears to be dominantly inherited, affects children and young adults, and may also be associated with leiomyomatosis of other viscera. Previously reported cases and possible aetiologies are reviewed, and evidence that this association represents a new variant of Alport's syndrome is discussed.

show abstract

Section: Discussionmentioning

confidence: 80%

Familial oesophageal leiomyomatosis and nephropathy

et al. 1992

View full text Add to dashboard Cite

show abstract

“…In man, the hypertrophy is also confined to the smooth muscle layer, mainly the inner circular layer. In many cases, a transmural lymphocytic and/or eosinophilic infiltration with involvement of the Auerbach's plexi is also present (Sloper 1954;Penka et al 1977;Iyer et al 1986;Legius et al 1990). In other reports of human cases, the intramural nerve plexuses appeared normal, although plexus nerve cells were sometimes reduced in numbers (Zeller et al 1979;Kreczy et al 1990;Legius et al 1990).…”

Section: Discussionmentioning

confidence: 99%

Idiopathic muscular hypertrophy of the oesophagus in the horse: a retrospective study of 31 cases

Benders

Kroeze

Kolk

2004

Equine Veterinary Journal

View full text Add to dashboard Cite

Reasons for performing study:To present the first report of a case series concerning equine idiopathic muscular hypertrophy of the oesophagus (IMHO). Objectives: To investigate the clinical and pathological features of the disorder. Methods: The medical records of 31 horses suffering from the disorder were reviewed retrospectively. In all these animals the diagnosis was confirmed at post mortem examination. Results: The median age of the affected horses was 12.5 ± 5.6 years (range 1-26) without sex or breed predilection. Only 2 out of 31 horses showed clinical signs associated with oesophageal dysfunction, indicating that the muscular hypertrophy was rather a coincidental post mortem finding. Histology revealed thickening of the distal portion of the oesophagus mainly involving the circular layer of the tunica muscularis without fibrosis or inflammation. In 8 cases, the disorder was seen in concurrence with idiopathic hypertrophy of the tunica muscularis of various other parts of the gastrointestinal tract. Conclusions: In the majority of patients, IMHO was a coincidental finding at post mortem examination usually confined to the smooth, circular muscle layer of the tunica muscularis externa. Potential relevance: Further research is necessary to study the precise effect of IMHO on oesophageal function.

show abstract

“…In a review of the English literature [3, 4, 5, 6, 7, 8, 9, 10, 11, 12]more than 20 case reports were found of esophageal leiomyomatosis and idiopathic muscular hypertrophy of the esophagus. Subjects ranged in age from 17 to 82 years and were more frequently men.…”

Section: Discussionmentioning

confidence: 99%

Esophageal Leiomyomatosis in a Woman with a History of Vulvar Leiomyoma and Barrett’s Esophagus

Compagnoni

Talamonti²,

Joob³

et al. 2000

Dig Surg

View full text Add to dashboard Cite

Background: The diagnosis and treatment of esophageal pathology remains a challenge despite advances in preoperative endoscopy, radiographic staging, and perioperative care. Case Report: In this article, we present an interesting case of esophageal leiomyomatosis in a woman with a history of vulvar leiomyoma and Barrett’s esophagus. This paper represents the first reported simultaneous occurrence of these three pathologic entities in the English literature. Conclusions: The clinical presentation and characteristic pathologic findings in patients with esophageal leiomyomatosis are reviewed. Diagnostic and therapeutic approaches to esophageal masses are discussed including the indications for esophageal resection.

show abstract

Diffuse muscular hypertrophy of esophagus

Cited by 17 publications

References 5 publications

Familial oesophageal leiomyomatosis and nephropathy

Familial oesophageal leiomyomatosis and nephropathy

Idiopathic muscular hypertrophy of the oesophagus in the horse: a retrospective study of 31 cases

Esophageal Leiomyomatosis in a Woman with a History of Vulvar Leiomyoma and Barrett’s Esophagus

Contact Info

Product

Resources

About