Dismenorrea. Una revisión multidisciplinaria en el contexto de la medicina basada en la evidencia

Abarca, L.I.; Molero, José; Casimiro, C.

doi:10.1016/s1134-2072(06)71400-1

Cited by 4 publications

(6 citation statements)

References 45 publications

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“…The sustained nociceptive stimuli that can cause hyperirritability of the MTrPs of a muscle can cause the secretion of sensitizing substances such as PGs, among others, which lower the activation threshold of the neuron such that they sensitize nociceptors [ 36 , 152 ]. These are PGs, which are also considered the main cause of the etiopathogenesis of menstrual pain [ 5 , 9 , 10 , 11 , 12 , 80 ].…”

Section: Discussionmentioning

confidence: 99%

“…Dysmenorrhea is considered one of the most prevalent gynecological disorders in women of reproductive age [ 1 , 2 ], reaching prevalence rates of up to 97% of the population in some locations [ 3 ], and ranging in Spain from 61.9% to 84.7% of the population [ 4 , 5 ]. Dysmenorrhea can be classified as secondary dysmenorrhea when pathological causes of the uterus or pelvis are responsible for the pain, the most frequent being endometriosis and pelvic inflammatory disorders, in addition to other causes, such as adenomyosis, uterine fibroids (myomas) or polyps, ovarian cysts, uterine malformations, pelvic adhesions, cervical stenosis of the uterus, pelvic congestion syndrome, and irritable bowel syndrome [ 1 , 6 , 7 , 8 ].…”

Section: Introductionmentioning

confidence: 99%

“…The etiology of PD is characterized by an increase in the synthesis and release of PGs, which cause hypercontractability of the myometrium with respect to the normal dynamics of the menstrual uterus. Hypercontractibility causes uterine ischemia and hypoxia and, consequently, an accumulation of metabolites and acids, which are also responsible for pain [ 5 , 9 , 10 , 11 , 12 ]. This increased contractility is mediated by the local release of nociceptive substances in response to hormonal stimuli.…”

Section: Introductionmentioning

confidence: 99%

“…This increased contractility is mediated by the local release of nociceptive substances in response to hormonal stimuli. There is also a sensitization of nerve endings to nociceptive stimuli [ 5 ]. The increased volume of menstrual flow due to ovulatory cycles increases the release of uterine PGs, which stimulates uterine contraction, increasing the tone, frequency, and intensity of contractions and causing colic-like pain [ 5 ].…”

Section: Introductionmentioning

confidence: 99%

“…There is also a sensitization of nerve endings to nociceptive stimuli [ 5 ]. The increased volume of menstrual flow due to ovulatory cycles increases the release of uterine PGs, which stimulates uterine contraction, increasing the tone, frequency, and intensity of contractions and causing colic-like pain [ 5 ]. However, Akman et al, found that in young women with PD, there are no differences in pain regardless of whether the cycles are ovulatory or anovulatory [ 13 ].…”

Section: Introductionmentioning

confidence: 99%

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Myofascial Pain Syndrome in Women with Primary Dysmenorrhea: A Case-Control Study

et al. 2022

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There is limited information on myofascial trigger points (MTrPs) and specific symptoms of chronic pelvic pain and, more specifically, dysmenorrhea. The objective of this study was to determine whether patients suffering from primary dysmenorrhea present alterations in mechanosensitivity and pain patterns, and greater presence of MTrPs in the abdominal and pelvic floor muscles. A case-control study was carried out with a total sample of 84 participants distributed based on primary dysmenorrhea and contraceptive treatment. The sample was divided into four groups each comprising 21 women. Data on pain, quality of life, and productivity and work absenteeism were collected; three assessments were made in different phases of the menstrual cycle, to report data on pressure pain threshold, MTrP presence, and referred pain areas. One-way ANOVA tests showed statistically significant differences (p < 0.01) between the groups, for the Physical Health domain and the total score of the SF-12 questionnaire, and for all the domains of the McGill questionnaire; but no significant differences were found in the data from the WPAI-GH questionnaire. Statistically significant data (p < 0.01) were found for mechanosensitivity in the abdominal area and limbs, but not for the lumbar assessment, within the group, with very few significant intergroup differences. The frequency of active MTrPs is higher in the groups of women with primary dysmenorrhea and during the menstrual phase, with the prevalence of myofascial trigger points of the iliococcygeus muscle being especially high in all examination groups (>50%) and higher than 70% in women with primary dysmenorrhea, in the menstrual phase, and the internal obturator muscle (100%) in the menstrual phase. Referred pain areas of the pelvic floor muscles increase in women with primary dysmenorrhea.

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