“…The transient otoacoustic emission intensity increase induced by sound stimulation can be established as a sensitive indicator of the changes in metabolism and operation of OHCs. This assumption has already been highlighted by Kiss et al, who studied the changes in human otoacoustic emission intensity after a 3 minutes exposure by wide-band noise and pure tone [13]. They documented a distortion product OAE (DPOAE) intensity increase also at low and high frequencies (500, 625, 781, 1000, 3187, 4000, and 5031 Hz) and a decrease at medium frequencies (1250, 1593, 2000, and 2531 Hz).…”