Distribution of ABO blood groups, G6PD deficiency, and abnormal haemoglobins in leprosy.

Saha, N.; Wong, Hee-Kit; Banerjee, Bhaswati; Wong, Morales

doi:10.1136/jmg.8.3.315

Cited by 10 publications

(6 citation statements)

References 8 publications

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“…This finding agrees with other previous works. For instance, a study in China on 459 Chinese leprosy patients and 15 261 healthy individuals found non-significant difference between leprosy patients and healthy controls [22]. In the same way, studies conducted in Bangladesh and India [23,24] are also consistent with the present study.…”

Section: Abo Blood Groupsupporting

confidence: 80%

Haptoglobin polymorphism, plasma haptoglobin level and ABO blood group in leprosy patients

Seboka¹,

Bekele²,

Nigussie³

et al. 2017

JCLM

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Section: Abo Blood Groupsupporting

confidence: 80%

Haptoglobin polymorphism, plasma haptoglobin level and ABO blood group in leprosy patients

Seboka¹,

Bekele²,

Nigussie³

et al. 2017

JCLM

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“…This finding is consistent with other previous studies. [1,2] However, Saha, et al (1973) [3] and Nydegger, et al (2003) [4] found that the O and B blood groups are predominant in patients with MI and may play a role in the pathogenesis of MI. Despite this, one cannot deny the potential importance of genetic factors in pathogenesis of MI.…”

Section: Abo Blood Groups and Myocardial Infarction Among Palestiniansmentioning

confidence: 99%

ABO blood groups and myocardial infarction among Palestinians

Skaik

2009

Ann Card Anaesth

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“…There is convincing evidence that ABO(H) blood groups are associated with thrombotic risk, resulting, in part, from higher levels of von Willebrand factor (VWF) in those with blood groups other than O [2,3]. A role for Lewis/Secretor status in thrombosis has also been reported, but is more controversial [4][5][6][7][8][9][10], as is the link between Lewis or Secretor and VWF levels [11][12][13][14]. In addition to a potential link with thrombosis, isomers of the Lewis antigens play a role in tissue differentiation, inflammation [15], and infection risk [16], in particular functioning as a ligand for selectins [15].…”

Section: Introductionmentioning

confidence: 99%

The influence of maternal Lewis, Secretor and ABO(H) blood groups on fetal growth restriction

Clark

Greer

2011

Journal of Thrombosis and Haemostasis

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Summary. Background: Fetal growth restriction (FGR) is associated with thrombosis of the placenta and an increased risk of subsequent vascular disease in the mother and fetus. The products of interactions between ABO(H), Lewis and Secretor genes are also associated with thrombosis and vascular disease risk. Objectives/Methods: A prospective case–control study of mothers with a severe FGR pregnancy (cases, n = 128; controls, n = 288) was performed to determine whether FGR is associated with particular maternal blood groups. Results: No association with ABO(H) status was observed, but FGR was more common in maternal secretors (odds ratio [OR] 1.70, 95% confidence interval [CI] 1.08–2.69) and consequently in those mothers expressing Le(b) on their red cells (OR 1.80, 95% CI 1.15–2.83), with a reduced risk in non‐secretors and those expressing Le(a). Given the association between blood groups and both activated protein C resistance (APCR) and von Willebrand factor (VWF) levels, post hoc pilot studies on first‐trimester APCR and VWF antigen levels and blood group genotypes were performed. No relationship with Lewis or Secretor was observed. Despite this, lower first‐trimester VWF levels were observed in pregnancies subsequently complicated by FGR. Conclusions: This is the first study reporting a relationship between maternal Secretor/Lewis status and FGR. A link between blood groups and FGR is plausible, as both are associated with cardiovascular disease. We observed no relationship between Lewis/Secretor status and VWF or APCR, but this should be confirmed in a larger study. Thus, the mechanism whereby Secretor and/or Lewis influences FGR is unknown.

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Distribution of ABO blood groups, G6PD deficiency, and abnormal haemoglobins in leprosy.

Cited by 10 publications

References 8 publications

Haptoglobin polymorphism, plasma haptoglobin level and ABO blood group in leprosy patients

Haptoglobin polymorphism, plasma haptoglobin level and ABO blood group in leprosy patients

ABO blood groups and myocardial infarction among Palestinians

The influence of maternal Lewis, Secretor and ABO(H) blood groups on fetal growth restriction

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