Do I Look Gawky? The Association between Pubertal Asynchrony and Peer Victimization

Natsuaki, Misaki N.; Stepanyan, Sofia T.; Neiderhiser, Jenaè M.; Shaw, Daniel S.; Ganiban, Jody M.; Reiss, David; Leve, Leslie D.

doi:10.3390/children8090794

Cited by 5 publications

(2 citation statements)

References 49 publications

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“…One possible explanation could be that the lack of coherence between different aspects of pubertal changes (eg, skin changes with lower height or without voice change) creates confusion or stress in male youth who feel distressed about their body development 40 ; some of the distress may be expressed as pain. However, asynchronous pubertal development was found to be associated with lower risk of depressive symptoms 58 and peer victimization 47 in boys. It is possible that inadequate alignment of body growth reflects problems associated with less coordinated biological maturation, which may increase the risk of pain.…”

Section: Discussionmentioning

confidence: 91%

Pubertal development and pain incidence and characteristics in children: a 1-year prospective cohort study of a national sample

Lopez

Gupta

et al. 2023

Pain

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Sex differences in pain become apparent during puberty. However, the influence of key pubertal characteristics and pubertal hormones on pain is largely unknown. We examined the prospective associations between self-reported and hormone-indicated pubertal characteristics and pain incidence and severity in 10- to 11-year-old pain-free youth in the Adolescent Brain Cognitive Development (ABCD) Study over 1 year. Puberty was measured at baseline and follow-up with self-report (Pubertal Development Scale [PDS]) and hormonal assessment (salivary dehydroepiandrosterone [DHEA], testosterone, and estradiol). Pain status (yes/no), intensity, and interference (0-10 numerical rating scale) in the past month were self-reported at follow-up. Pubertal maturity, progression, and asynchrony were examined in relation to pain onset and severity through confounder-adjusted generalized estimating equations modified Poisson and linear mixed regression models. Among 6631 pain-free youth at baseline, 1-year incident pain was 30.7%. In both sexes, higher PDS scores were associated with greater risk of pain onset (relative risk [RR] = 1.10 to 1.27, Ps < 0.01). In boys, higher PDS item variance was associated with greater pain incidence (RR = 1.11, 95% CI, 1.03-1.20) and interference (beta = 0.40, 95% CI, 0.03-0.76); higher PDS overall and gonadal scores were associated with higher pain intensity (Ps < 0.05). Associations with hormones were seen in boys only, with each 10-fold higher testosterone levels associated with a 40% lower risk of pain incidence (95% CI, −55% to −22%) and 1.30-point lower (95% CI, −2.12 to −0.48) pain intensity, and higher DHEA levels were associated with lower pain intensity (P = 0.020). Relationships between pubertal development and pain in peripubertal adolescents are sex specific and puberty measurement specific and warrant further investigation.

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Section: Discussionmentioning

confidence: 91%

Pubertal development and pain incidence and characteristics in children: a 1-year prospective cohort study of a national sample

Lopez

Gupta

et al. 2023

Pain

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“…We estimate the overall retention rate for both cohorts at age 11 to be at 75%. So far, recent work using later waves of data (ages 7-11) has shown no evidence of a systematic pattern in missingness (Ganiban et al, 2021;Natsuaki et al, 2021) with a few minor exceptions (e.g., openness was higher for families with missing data; Cioffi, Griffin, et al, 2021); the Missing Completely at Random [MCAR] assumption did not hold, but data were consistent with the Missing at Random [MAR] assumption (Austerberry et al, 2021).…”

Section: Timeline Of Egds Assessments and Retention Rates Through Age 11mentioning

confidence: 99%

Parenting in the Context of the Child: Genetic and Social Processes

Reiss

Ganiban

Leve³

et al. 2022

Monographs Society Res Child

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The focus on the role of parenting in child development has a long‐standing history. When measures of parenting precede changes in child development, researchers typically infer a causal role of parenting practices and attitudes on child development. However, this research is usually conducted with parents raising their own biological offspring. Such research designs cannot account for the effects of genes that are common to parents and children, nor for genetically influenced traits in children that influence how they are parented and how parenting affects them. The aim of this monograph is to provide a clearer view of parenting by synthesizing findings from the Early Growth and Development Study (EGDS).EGDS is a longitudinal study of adopted children, their birth parents, and their rearing parents studied across infancy and childhood. Families (N = 561) were recruited in the United States through adoption agencies between 2000 and 2010. Data collection began when adoptees were 9 months old (males = 57.2%; White 54.5%, Black 13.2%, Hispanic/Latinx 13.4%, Multiracial 17.8%, other 1.1%). The median child age at adoption placement was 2 days (M = 5.58, SD = 11.32). Adoptive parents were predominantly in their 30s, White, and coming from upper‐middle‐ or upper‐class backgrounds with high educational attainment (a mode at 4‐year college or graduate degree). Most adoptive parents were heterosexual couples, and were married at the beginning of the project. The birth parent sample was more racially and ethnically diverse, but the majority (70%) were White. At the beginning of the study, most birth mothers and fathers were in their 20s, with a mode of educational attainment at high school degree, and few of them were married. We have been following these family members over time, assessing their genetic influences, prenatal environment, rearing environment, and child development.Controlling for effects of genes common to parents and children, we confirmed some previously reported associations between parenting, parent psychopathology, and marital adjustment in relation to child problematic and prosocial behavior. We also observed effects of childrenʼs heritable characteristics, characteristics thought to be transmitted from parent to child by genetic means, on their parents and how those effects contributed to subsequent child development. For example, we found that genetically influenced child impulsivity and social withdrawal both elicited harsh parenting, whereas a genetically influenced sunny disposition elicited parental warmth. We found numerous instances of children's genetically influenced characteristics that enhanced positive parental influences on child development or that protected them from harsh parenting. Integrating our findings, we propose a new, genetically informed process model of parenting. We posit that parents implicitly or explicitly detect genetically influenced liabilities and assets in their children. We also suggest future research into factors such as marital adjustment, that favor parents responding with appropriate protection or enhancement. Our findings illustrate a productive use of genetic information in prevention research: helping parents respond effectively to a profile of child strengths and challenges rather than using genetic information simply to identify some children unresponsive to current preventive interventions.

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Appearance-Related Victimization and Pubertal Asynchrony: Identifying Sex-Specific Vulnerabilities

Bigelow,

Chaku

2024

J. Youth Adolescence

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Do I Look Gawky? The Association between Pubertal Asynchrony and Peer Victimization

Cited by 5 publications

References 49 publications

Pubertal development and pain incidence and characteristics in children: a 1-year prospective cohort study of a national sample

Pubertal development and pain incidence and characteristics in children: a 1-year prospective cohort study of a national sample

Parenting in the Context of the Child: Genetic and Social Processes

Appearance-Related Victimization and Pubertal Asynchrony: Identifying Sex-Specific Vulnerabilities

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