Does Air Pollution Trigger Infant Mortality in Western Europe? A Case-Crossover Study.

Scheers, Hans; Mwalili, Samuel; Faes, Christel; Nemery, Benoît; Nawrot, Tim S.

doi:10.1289/isee.2011.00864

Cited by 7 publications

(12 citation statements)

References 31 publications

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“…Therefore, all participants were assigned daily exposures from the same measurement site, giving us the temporal signal only. This could be a limitation, however, we don't believe that this influenced the results because evidence shows that temporal differences are much more determining than spatial differences in air pollution [37].…”

Section: Discussionmentioning

confidence: 89%

“…Studies show a very good correlation among the day-to-day changes in measurement stations and personal exposure [35,36]. We also know from previous data that spatial variability in PM10 is less important than temporal variability, as the latter is largely caused by weather changes [37]. However, added personal exposure based on commute and travelling was not evaluated due to ethical and privacy reasons and could therefore influence the results.…”

Section: Discussionmentioning

confidence: 99%

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The impact of acute air pollution fluctuations on bronchiectasis pulmonary exacerbation: a case-crossover analysis

et al. 2018

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In bronchiectasis, exacerbations are believed to be triggered by infectious agents, but often no pathogen can be identified. We hypothesised that acute air pollution exposure may be associated with bronchiectasis exacerbations.We combined a case-crossover design with distributed lag models in an observational record linkage study. Patients were recruited from a specialist bronchiectasis clinic at Ninewells Hospital, Dundee, UK.We recruited 432 patients with clinically confirmed bronchiectasis, as diagnosed by high-resolution computed tomography. After excluding days with missing air pollution data, the final model for particles with a 50% cut-off aerodynamic diameter of 10 µm (PM) was based on 6741 exacerbations from 430 patients and for nitrogen dioxide (NO) it included 6248 exacerbations from 426 patients. For each 10 µg·m³ increase in PM and NO, the risk of having an exacerbation that same day increased significantly by 4.5% (95% CI 0.9-8.3) and 3.2% (95% CI 0.7-5.8) respectively. The overall (lag zero to four) increase in risk of exacerbation for a 10 μg·m increase in air pollutant concentration was 11.2% (95% CI 6.0-16.8) for PM and 4.7% (95% CI 0.1-9.5) for NO Subanalysis showed higher relative risks during spring (PM 1.198 (95% CI 1.102-1.303), NO 1.146 (95% CI 1.035-1.268)) and summer (PM 2.142 (95% CI 1.785-2.570), NO 1.352 (95% CI 1.140-1.602)) when outdoor air pollution exposure would be expected to be highest.In conclusion, acute air pollution fluctuations are associated with increased exacerbation risk in bronchiectasis.

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Section: Discussionmentioning

confidence: 89%

Section: Discussionmentioning

confidence: 99%

The impact of acute air pollution fluctuations on bronchiectasis pulmonary exacerbation: a case-crossover analysis

et al. 2018

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“…The estimates of Pope et al (2002) were applied to the European data by CAF E and W HO to calculate that the short-term and long-term exposures were together responsible for 370 000 premature deaths in 2000 in Europe (WHO 2004a). The infant mortality risk has been studied by for example by Currie and Neidell (2004), Chay and Greenstone (2003)and Scheers et al (2011). WHO has summarized that, taken all types of deaths together, urban outdoor air pollution causes 1.3 million deaths worldwide per year (WHO 2011)…”

Section: The Positive Check -Recent Evidencementioning

confidence: 99%

Optimal Pollution, Optimal Population, and Sustainability

Lehmijoki

2013

Dynamic Modeling and Econometrics in Economics and Finance

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This paper develops a long-run consumer optimization model with endogenous pollution and endogenous population. The positive check increases mortality if pollution increases. The optimal path is sustainable if it provides non-decreasing consumption for a non-decreasing population. As usually, optimality and sustainability may conflict; with population endogenous to pollution, this conflict may ultimately lead the human species toward self-imposed extinction. Not even technical progress can warrant sustainability.

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“…1 Prenatal exposure to environ mental toxicants might cause developmental changes in fetuses, affecting the structure, physiology, and function of organ systems. 2,3 Prenatal exposure to toxicants has been linked to fetal growth restriction, adverse birth outcomes, infant mortality, [4][5][6][7] and longterm health challenges that persist over the life course. 2,8,9 Vanadium, an abundant transition metal, 10 is released into the atmosphere from industrial activities 11 and the burning of crude oil, coal, and heavy oils.…”

Section: Introductionmentioning

confidence: 99%

Effects of trimester-specific exposure to vanadium on ultrasound measures of fetal growth and birth size: a longitudinal prospective prenatal cohort study

Yang

Zheng

et al. 2018

The Lancet Planetary Health

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Background Identification of windows of heightened vulnerability to environmental factors has substantial public health implications. Prenatal exposure to vanadium has been linked to adverse birth outcomes; however, critical windows for such exposure during fetal growth remain unknown. We aimed to assess trimester-specific associations of vanadium exposure with ultrasound measures of fetal growth and birth size in a Chinese longitudinal cohort. Methods The present study was embedded in our ongoing prospective prenatal cohort study at the Wuhan Women and Children Medical Care Center (Wuhan, Hubei, China). Pregnant women were eligible for inclusion if they provided signed informed consent and were less than 16 weeks pregnant with a single gestation, and agreed to take in-person interviews, undergo ultrasound examinations, and provide blood and urine samples. We collected urine samples and measured urinary vanadium concentrations using inductively coupled plasma mass spectrometry. We calculated SD scores for ultrasound-measured biparietal diameter, head circumference, occipitofrontal diameter, abdominal circumference, femur length, and estimated fetal weight at 16, 24, and 31 weeks of gestation. We applied linear regressions with generalised estimating equations to estimate associations of urinary vanadium concentrations in each trimester with ultrasound-measured fetal growth parameters or neonatal size at birth. Findings As of Oct 12, 2016, we recruited 3075 women who were non-smokers and non-drinkers during pregnancy, provided up to three urine samples during the first, second, and third trimesters, and gave birth to live singletons without birth defects. We excluded women who did not provide information on ultrasound measurements (n=20) or who only had one ultrasound measurement of fetal crown-rump length at the first trimester (n=14). We excluded another 16 women because they had missing values for confounding variables, leaving 3025 women retained in the study. Every doubling of urinary vanadium concentration in the first trimester was associated with a significant increase in femur length (adjusted percentage change 6•4%, 95% CI 0•7 to 12•1) at 16 weeks of gestation and reductions in biparietal diameter (-4•2%, −8•2 to-0•1), head circumference (−6•0%,-10•1 to −1•9), occipitofrontal diameter (−5•7%, −9•9 to −1•5), and abdominal circumference (−5•3%, −9•4 to −1•2) at 31 weeks of gestation. Every doubling of urinary vanadium concentration in the second trimester was significantly associated with reductions in SD scores for head circumference (-7•2%, −14•1 to −0•3) and abdominal circumference (−6•9%, −13•8 to −0•1) at 31 weeks of gestation. The highest quartile of urinary vanadium concentration (>1•18 μg/L) in the first trimester, when compared with the lowest quartile (≤0•60 μg/L), was associated with a mean decrease in birthweight of 12•6 g (95% CI 2•5-22•8; p trend =0•0055) and a mean decrease in ponderal index of 0•07 kg/m³ (0•01-0•12; p trend =0•0053). Moreover, newborns with restricted birth size had higher ...

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Does Air Pollution Trigger Infant Mortality in Western Europe? A Case-Crossover Study.

Cited by 7 publications

References 31 publications

The impact of acute air pollution fluctuations on bronchiectasis pulmonary exacerbation: a case-crossover analysis

The impact of acute air pollution fluctuations on bronchiectasis pulmonary exacerbation: a case-crossover analysis

Optimal Pollution, Optimal Population, and Sustainability

Effects of trimester-specific exposure to vanadium on ultrasound measures of fetal growth and birth size: a longitudinal prospective prenatal cohort study

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