Dynamics of a liquid lamella resulting from the impact of a water drop on a small target

Рожков, А. Н.; Prunet-Foch, B.; Vignes-Adler, M.

doi:10.1098/rspa.2004.1293

Cited by 58 publications

(103 citation statements)

References 13 publications

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“…The symmetry breaking apparent on the distorted rim (on can tell the direction of the acceleration by looking at still pictures) where thicker portions of the rim are "left behind" thinner ones while accelerated at constant force, 12 together with the fair quantitative agreement between the observations and Eq. (4) both tend to support the relevance of the Rayleigh-Taylor scenario: The driving force is inertia (acceleration), and the mode selection is made by capillarity.…”

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confidence: 50%

The destabilization of an initially thick liquid sheet edge

Lhuissier

Villermaux

2011

Physics of Fluids

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International audienceBy forcing the sudden dewetting of a free soap film attached on one edge to a straight solid wire, we study the recession and subsequent destabilization of its free edge. The newly formed rim bordering the sheet is initially thicker than the film to which it is attached, because of the Plateau border preexisting on the wire. The initial condition is thus that of an immobile massive toroidal rim connected to a thin liquid film of thickness h. The terminal Taylor-Culick receding velocity V = sqrt(2 sigma/rho h), where sigma and rho are the liquid surface tension and density, respectively, is only reached after a transient acceleration period which promotes the rim destabilization. The selected wavelength and associated growth time coincide with those of an inertial instability driven by surface tension

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confidence: 50%

The destabilization of an initially thick liquid sheet edge

Lhuissier

Villermaux

2011

Physics of Fluids

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“…A similar scenario has been observed when a viscous drop impacts a thin layer of liquid with a lower surface tension [22]. For pure water (no oil droplets), no perforation occurs and the sheet uniquely disintegrates through the destabilization of the external rim into drops [12]. Thanks to the dye, the modulations of the sheet thick- ness are visualized.…”

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confidence: 77%

“…But, to the best of our knowledge, those conditions have never been confronted to robust experimental facts. To unambiguously clarify the physical mechanisms at play, rationale experiments on individual perforation events are therefore required.In this Letter, we investigate the perforation mechanisms of an emulsion-based free liquid sheet issued from a single-drop experiment; resulting from the impact of one drop of fluid onto a small target [12][13][14]. During the sheet expansion, holes nucleate and grow.…”

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confidence: 99%

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Bursting of Dilute Emulsion-Based Liquid Sheets Driven by a Marangoni Effect

2015

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We study the destabilization mechanism of thin liquid sheets expanding in air and show that dilute oil-in-water emulsion-based sheets disintegrate through the nucleation and growth of holes that perforate the sheet. The velocity and thickness fields of the sheet outside the holes are not perturbed by holes and hole opening follows a Taylor-Culick law. We find that a pre-hole, which widens and thins out the sheet with time, systematically precedes the hole nucleation. The growth dynamics of the pre-hole follows the law theoretically predicted for a liquid spreading on another liquid of higher surface tension due to Marangoni stresses. Classical Marangoni spreading experiments quantitatively corroborate our findings.The destabilization of free liquid films is of great importance for aerosol dispersions, and is involved in many practical situations [1,2] [6], and dilute oil-in-water emulsions [7]. However, despite its relative ubiquity, the bursting of liquid sheets through perforation events have not yet been carefully investigated nor modeled. The occurrence of perforation events in a spray directly decreases the fraction of small drops issued from the spray [8] as illustrated in the case of dilute emulsions, which are prone to induce the bursting of liquid sheets [7]. Dilute emulsions are also recognized as anti-drift adjuvants [8][9][10][11] and therefore commonly used as carrier fluids for pesticides delivery. Controlling the perforation processes would therefore allow one to finely tune the size distribution of drops issued from sprays, a goal of uttermost importance in many industrial processes. In this optics, a physical description of the mechanisms at play in perforation processes is desired. The commonly invoked conditions for perforation include a dewetting of inclusions by the fluid and an inclusion diameter equal to, or larger than, the thickness of the liquid sheet, so that inclusions cause perforation by puncturing both interfaces of the sheet [3]. But, to the best of our knowledge, those conditions have never been confronted to robust experimental facts. To unambiguously clarify the physical mechanisms at play, rationale experiments on individual perforation events are therefore required.In this Letter, we investigate the perforation mechanisms of an emulsion-based free liquid sheet issued from a single-drop experiment; resulting from the impact of one drop of fluid onto a small target [12][13][14]. During the sheet expansion, holes nucleate and grow. We show that each perforation event is preceded by the formation of a pre-hole that thins out the sheet and widens with time. We demonstrate that the pre-hole growth is governed by a Marangoni effect. The entry of emulsion oil droplets at the air/water interface leads to a spreading of the oil due to a surface tension gradient stress. This stress is counterbalanced by a viscous stress that drags the subsurface fluid, whose flow causes a local film thinning which ultimately lead to the rupture of the film. We show that the growth kinetics of pre-holes ...

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“…In recent researches reported that, in 3D environments, E-cadherin deficiency indeed led to a loss of intercellular adhesion and triggered tumor cell invasion by matrix MMP-2 and MMP-9 driven matrix degradation. Surface tension at the tumor-tissue interface, have been extensively studied in the field of fluid dynamics (44,45,46,47). Promoting tumor cell adhesion and thus increasing the tumor surface tension can induce cellular cohesiveness and decreasing of the invasiveness.…”

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confidence: 99%

Invasion Mechanisms of Bladder Cancer: A Molecular Review

Narter¹,

Sabuncu²

2016

jus

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ÖZBladder cancer (BC) is a very common cancer and it has high mortality rates, especially in late stages. BC is considered as a different disease in various stages and grades. Non-muscle-invasive BC and muscle-invasive BC have different properties. There are some prognostic factors for progression and recurrence rates of BC and we have some risk assessment methods. These factors are based on clinical findings and histopathological properties. We do not know which factors and molecular processes are effective in the invasion mechanism. In this review, we summarized possible invasion mechanisms of BC. KeywordsBladder cancer, invasion, metastasis, molecularMesane kanseri farklı evrelerde farklı birer hastalık gibi kabul ve tedavi edilir. Her evre tekrarlama ve ilerleme açısından farklı riskler taşır. Günümüzde bu tekrarlama ve ilerleme risklerini gösteren değişik hesaplama sistemleri tanımlansa da altta yatan moleküler, genetik ve çevresel faktörler net olarak gösterilememiştir. Mevcut risk hesaplama sistemleri klinik, makroskopik ve patolojik değerlendirme temellerine oturmaktadır. Derlemede özetlemeye çalıştığımız olası invazyon mekanizmaları aydınlatıldıkça prognostik yeni moleküler faktörlerin tanımlanması, hatta bu temelde yeni tedavi metodlarının geliştirilmesi mümkün olacaktır. Anahtar KelimelerMesane kanseri, invazyon, metastaz, moleküler IntroductionUrothelial tumors are classified as papillary and nonpapillary (solid) tumors. Histopathologically, more than 90% of bladder cancers (BCs) are urothelial (transitional) cell carcinomas (UCC or TCC). At the time of diagnosis, about 75% of BCs are non-muscle-invasive bladder cancer (NMIBC) and remaining 25% are muscle-invasive bladder cancer (MIBC). Phenotypes and genotypes of these cancers in various stages are different. In this context, 30% to 50% of NMIBCs recur after transurethral resection of the primary tumor, and 10% to 20% progress to MIBC. Many of high-grade carcinomas (including carcinoma in situ) invade the bladder wall and may metastasize to other sites. Additionally, nearly 50% of patients with MIBCs have occult distant metastases at the time of diagnosis (1,2). Recently, in the literature, there are many studies in which molecular, genetic and physical factors have been investigated in the progression and recurrence of the BC but it has not been fully outlined. All these factors affect the process of metastasis, invasion and resistance to therapy. Generally, major characteristics of malignant cancer cells are defined as self-sufficiency in growth signals, insensitivity to anti-growth signals, tissue invasion and metastasis, limitless replicative potential, sustained angiogenesis, and evading apoptosis. Additionally, tumor metastasis includes detachment, migration, invasion, intravasation, anoikis, evasion, and extravasation steps. In this review, we aimed to summarize the possible genetic, molecular and physical factors that are responsible for the progression and recurrence. Epithelial-Mesenchymal Transition and PlasticityMechanism of the...

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Dynamics of a liquid lamella resulting from the impact of a water drop on a small target

Cited by 58 publications

References 13 publications

The destabilization of an initially thick liquid sheet edge

The destabilization of an initially thick liquid sheet edge

Bursting of Dilute Emulsion-Based Liquid Sheets Driven by a Marangoni Effect

Invasion Mechanisms of Bladder Cancer: A Molecular Review

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