Echocardiographic-morphologic correlations in tricuspid atresia

Orie, Joseph; Anderson, Christine; Ettedgui, Jose A.; Zuberbuhler, James R.; Anderson, Robert H.

doi:10.1016/0735-1097(95)00250-8

Cited by 33 publications

(10 citation statements)

References 11 publications

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“…This is hardly surprising, since the morphological substrate for the atresia in almost all cases is absence of the entire right atrioventricular connection, including the tricuspid valve and its vestibule. The lesion is only very rarely due to an imperforate atrioventricular valve 22. Our dissections are unequivocal in showing the absence of the right atrioventricular connections.…”

Section: Discussionsupporting

confidence: 48%

Myoarchitecture and connective tissue in hearts with tricuspid atresia

Sánchez‐Quintana¹,

Climent²,

Ho³

et al. 1999

Heart

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Objective-To compare the atrial and ventricular myoarchitecture in the normal heart and the heart with tricuspid atresia, and to investigate changes in the three dimensional arrangement of collagen fibrils. Methods-Blunt dissection and cell maceration with scanning electron microscopy were used to study the architecture of the atrial and ventricular musculature and the arrangement of collagen fibrils in three specimens with tricuspid atresia and six normal human hearts. Results-There were significant modifications in the myoarchitecture of the right atrium and the left ventricle, both being noticeably hypertrophied. The middle layer of the ventricle in the abnormal hearts was thicker than in the normal hearts. The orientation of the superficial layer in the left ventricle in hearts with tricuspid atresia was irregular compared with the normal hearts. Scanning electron microscopy showed coarser endomysial sheaths and denser perimysial septa in hearts with tricuspid atresia than in normal hearts. Conclusions-The overall architecture of the muscle fibres and its connective tissue matrix in hearts with tricuspid atresia diVered from normal, probably reflecting modelling of the myocardium that is inherent to the malformation. This is in concordance with clinical observations showing deterioration in pump function of the dominant left ventricle from very early in life.

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Section: Discussionsupporting

confidence: 48%

Myoarchitecture and connective tissue in hearts with tricuspid atresia

Sánchez‐Quintana¹,

Climent²,

Ho³

et al. 1999

Heart

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show abstract

“…Some authors [78] elected to treat this bdimpleQ as a diminutive valve and apply cryoablation lesions to connect the coronary sinus with the bdimpleQ. However, the anatomic dissections performed by Orie et al [81] showed the AV node in very close proximity to the proposed cryoablation area. Our approach to patients with atrial reentry tachycardia and tricuspid atresia is to omit the cryoablation lesion that connects the coronary sinus with the presumed annulus of the diminutive tricuspid annulus.…”

Section: Surgical Managementmentioning

confidence: 97%

“…Patients with tricuspid atresia have a btricuspid dimpleQ in about a third of cases [81,82]. Some authors [78] elected to treat this bdimpleQ as a diminutive valve and apply cryoablation lesions to connect the coronary sinus with the bdimpleQ.…”

Section: Surgical Managementmentioning

confidence: 99%

Surgery for arrhythmias in children

Mavroudis

Deal

Backer

2004

International Journal of Cardiology

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“…The atretic tricuspid valve is usually a muscular, and rarely a fibrous, plate [30]. The atretic tricuspid valve is usually a muscular, and rarely a fibrous, plate [30].…”

Section: Tricuspid Atresiamentioning

confidence: 99%