Stroke Rehabilitation 2016
DOI: 10.1016/b978-0-323-17281-3.00023-x
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Edema Control

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Cited by 2 publications
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“…Poststroke hand oedema occurs in 37% of individuals who experience a chronic stroke and in up to 18.5% of individuals with acute stroke (Gebruers, Truijen, Engelborghs, & De Deyn, 2011;Leibovitz et al, 2007). Although the exact aetiology of poststroke hand oedema is still inconclusive, a few possible causes have been identified, including sympathetic vasomotor dysfunction and dysregulation of the autonomic nervous system caused by stroke (Artzberger & White, 2011;Hesse, Jahnke, Ehret, & Mauritz, 1995), venous congestion due to immobility, and dependent positioning (Artzberger & White, 2011;Geurts, Visschers, van Limbeek, & Ribbers, 2000). Vascular changes after stroke might also alter the mechanism of filtration and reabsorption of excessive amount of interstitial fluid in the vessels, which may also lead to hand oedema (Wang, Chen, Lan, Wong, & Lai, 2004;Wang, Yang, Liaw, & Wong, 2002).…”
Section: Introductionmentioning
confidence: 99%
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“…Poststroke hand oedema occurs in 37% of individuals who experience a chronic stroke and in up to 18.5% of individuals with acute stroke (Gebruers, Truijen, Engelborghs, & De Deyn, 2011;Leibovitz et al, 2007). Although the exact aetiology of poststroke hand oedema is still inconclusive, a few possible causes have been identified, including sympathetic vasomotor dysfunction and dysregulation of the autonomic nervous system caused by stroke (Artzberger & White, 2011;Hesse, Jahnke, Ehret, & Mauritz, 1995), venous congestion due to immobility, and dependent positioning (Artzberger & White, 2011;Geurts, Visschers, van Limbeek, & Ribbers, 2000). Vascular changes after stroke might also alter the mechanism of filtration and reabsorption of excessive amount of interstitial fluid in the vessels, which may also lead to hand oedema (Wang, Chen, Lan, Wong, & Lai, 2004;Wang, Yang, Liaw, & Wong, 2002).…”
Section: Introductionmentioning
confidence: 99%
“…Persistent hand oedema is correlated with pain and fibrosis of the tissue, which have negative effects on hand functions (Boomkamp-Koppen, Visser-Meily, Post, & Prevo, 2005;Geurts et al, 2000). The two most common outcome measures of hand oedema are circumferential measurements and volumetric measurement (Artzberger & White, 2011). The rehabilitation management of poststroke hand oedema includes electrical stimulation (Faghri, 1997;Pandyan, Powell, Futter, Granat, & Stott, 1996), compression therapy (Bell & Muller, 2013;Gustafsson, Walter, Bower, Slaughter, & Hoyle, 2014;Roper, Redford, & Tallis, 1999), orthosis (Bürge et al, 2008;Gracies et al, 2000;Kuppens, Pijlman, Hitters, & van Heugten, 2014), and mobilization (Dirette & Hinojosa, 1994;Giudice, 1990;Kim, Lee, & Sohng, 2014).…”
Section: Introductionmentioning
confidence: 99%