Effect of bidi cigarette smoking on interleukin-1β and 8 levels in chronic periodontitis patient

Nath, Sonia; Prakash, Jayant; Prajapati, Virendra Kumar; Sharma, Nitesh; Pulikkotil, Shaju Jacob

doi:10.4103/ijdr.ijdr_783_17

Indian J Dent Res

2020

DOI: 10.4103/ijdr.ijdr_783_17

|View full text |Cite

Effect of bidi cigarette smoking on interleukin-1β and 8 levels in chronic periodontitis patient

Sonia Nath

Jayant Prakash

Virendra Kumar Prajapati

et al.

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...

Citation Types

Supporting

Mentioning

Contrasting

Year Published

2024

Publication Types

Select...

Preprint1

Relationship

Self Cite0

Independent1

Authors

Journals

Cited by 1 publication

References 1 publication

Supporting

Mentioning

Contrasting

Order By: Relevance

A scope systematic review to clarify immune response to tobacco in Buerger

Ahmadi,

Khaghanzadeh,

Mirlohi

et al. 2024

Preprint

View full text Add to dashboard Cite

Background Progressive Thromboangiitis Obliterans (TAO) is a progressive and segmental inflammatory disorder known as Buerger's disease that affects the medium and small arteries of the upper limbs. The major risk factor for Buerger's disease is cigarettes and tobacco products, which can activate several inflammatory and pre-inflammatory parameters. Methods For this systematic review, based on the accepted criteria of Prisma, we searched from 2017 to 2022 in PubMed, Web of Science, science direct and followed the results of Google Scholar cytokines. We reviewed the literature on these smoking-related immune parameters in Buerger's disease as a potential treatment for this disease. Results In TOA patients, IL-17, RORγt, HMGB1 and RAGE mRNA expression are positively correlated with symptom severity. TLR family is associated with changes in the TOA population as a significant difference in TLR4 in the resting and acute phases. TLR9 may also be responsible for the secretion of IL-8 TNF involves in inflammation, muscle weakness, and pain in TOA. significant difference of IL-33 levels between TOA patients, healthy smokers and non-smokers of this cytokine. Conclusion Our results suggest that Buerger's disease can be considered an autoimmune disease caused by smoking. Buerger increase pro-inflammation markers in TOA and reduce angiogenetic factors. We recommend that pro-inflammatory cytokines should be considered in treatment and diagnosis programs and further research should focus on them.

show abstract

A scope systematic review to clarify immune response to tobacco in Buerger

Ahmadi,

Khaghanzadeh,

Mirlohi

et al. 2024

Preprint

View full text Add to dashboard Cite

show abstract

scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.

Contact Info

customersupport@researchsolutions.com

10624 S. Eastern Ave., Ste. A-614

Henderson, NV 89052, USA

This site is protected by reCAPTCHA and the Google Privacy Policy and Terms of Service apply.

Blog Terms and Conditions API Terms Privacy Policy Contact Cookie Preferences Do Not Sell or Share My Personal Information

Made with 💙 for researchers

Part of the Research Solutions Family.

Effect of bidi cigarette smoking on interleukin-1β and 8 levels in chronic periodontitis patient

Cited by 1 publication

References 1 publication

A scope systematic review to clarify immune response to tobacco in Buerger

A scope systematic review to clarify immune response to tobacco in Buerger

Contact Info

Product

Resources

About